(Q46856395)

English

Efficient execution of cell death in non-glycolytic cells requires the generation of ROS controlled by the activity of mitochondrial H+-ATP synthase

scientific article published on 16 December 2005

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scholarly article

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Europe PubMed Central

25 December 2017

http://europepmc.org/abstract/MED/16361271

Efficient execution of cell death in non-glycolytic cells requires the generation of ROS controlled by the activity of mitochondrial H+-ATP synthase (English)

1 reference

Europe PubMed Central

25 December 2017

http://europepmc.org/abstract/MED/16361271

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Isabel Fabregat

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Isabel Fabregat

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Gema Santamaría

1

1 reference

Europe PubMed Central

25 December 2017

http://europepmc.org/abstract/MED/16361271

Marta Martínez-Diez

2

1 reference

Europe PubMed Central

25 December 2017

http://europepmc.org/abstract/MED/16361271

José M Cuezva

4

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Europe PubMed Central

25 December 2017

http://europepmc.org/abstract/MED/16361271

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16 December 2005

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Europe PubMed Central

25 December 2017

http://europepmc.org/abstract/MED/16361271

1 reference

Europe PubMed Central

25 December 2017

http://europepmc.org/abstract/MED/16361271

27

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Europe PubMed Central

25 December 2017

http://europepmc.org/abstract/MED/16361271

925-935

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Europe PubMed Central

25 December 2017

http://europepmc.org/abstract/MED/16361271

5

1 reference

Europe PubMed Central

25 December 2017

http://europepmc.org/abstract/MED/16361271

Mitochondrial diseases in man and mouse

1 reference

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21 January 2018

The ATP synthase--a splendid molecular machine

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Organelle-specific initiation of cell death pathways

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Mechanisms of apoptosis avoidance in cancer.

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Multiple cell death pathways as regulators of tumour initiation and progression.

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Cell metabolism in the regulation of programmed cell death

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BAD and glucokinase reside in a mitochondrial complex that integrates glycolysis and apoptosis

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In self-defence: hexokinase promotes voltage-dependent anion channel closure and prevents mitochondria-mediated apoptotic cell death

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21 January 2018

AIF deficiency compromises oxidative phosphorylation

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Bcl-2 blocks apoptosis in cells lacking mitochondrial DNA.

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Lack of oxidative phosphorylation and low mitochondrial membrane potential decrease susceptibility to apoptosis and do not modulate the protective effect of Bcl-x(L) in osteosarcoma cells.

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Resistance of mitochondrial DNA-depleted cells against cell death: role of mitochondrial superoxide dismutase

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Mitochondrial respiratory chain inhibitors induce apoptosis.

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Mitochondria in eosinophils: functional role in apoptosis but not respiration

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The Mitochondrial F0F1-ATPase proton pump is required for function of the proapoptotic protein Bax in yeast and mammalian cells.

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Oligomycin, inhibitor of the F0 part of H+-ATP-synthase, suppresses the TNF-induced apoptosis.

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Role of oxidative phosphorylation in Bax toxicity.

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21 January 2018

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Alteration of the bioenergetic phenotype of mitochondria is a hallmark of breast, gastric, lung and oesophageal cancer.

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The bioenergetic signature of lung adenocarcinomas is a molecular marker of cancer diagnosis and prognosis

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21 January 2018

Proteomic changes in renal cancer and co-ordinate demonstration of both the glycolytic and mitochondrial aspects of the Warburg effect

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21 January 2018

Decrease of mitochondrial DNA content and energy metabolism in renal cell carcinoma.

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Alteration of the copy number and deletion of mitochondrial DNA in human hepatocellular carcinoma

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21 January 2018

Identification of tumor-associated proteins in oral tongue squamous cell carcinoma by proteomics.

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21 January 2018

Down-regulation of mitochondrial F1F0-ATP synthase in human colon cancer cells with induced 5-fluorouracil resistance

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21 January 2018

Activation of caspases occurs downstream from radical oxygen species production, Bcl-xL down-regulation, and early cytochrome C release in apoptosis induced by transforming growth factor beta in rat fetal hepatocytes

1 reference

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21 January 2018

Reactive oxygen species (ROS) mediates the mitochondrial-dependent apoptosis induced by transforming growth factor (beta) in fetal hepatocytes.

1 reference

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21 January 2018

Changes in intramitochondrial and cytosolic pH: early events that modulate caspase activation during apoptosis

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21 January 2018

Bafilomycins: a class of inhibitors of membrane ATPases from microorganisms, animal cells, and plant cells

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21 January 2018

Triggering caspase-independent cell death to combat cancer.

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21 January 2018

Mitochondria in cell death: novel targets for neuroprotection and cardioprotection

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21 January 2018

Stress-induced activation of the p53 tumor suppressor in leukemia cells and normal lymphocytes requires mitochondrial activity and reactive oxygen species.

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21 January 2018

Preservation of mitochondrial structure and function after Bid- or Bax-mediated cytochrome c release

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21 January 2018

The role of dynamin-related protein 1, a mediator of mitochondrial fission, in apoptosis

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21 January 2018

DRP-1-mediated mitochondrial fragmentation during EGL-1-induced cell death in C. elegans

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21 January 2018

Mitochondrial fusion and fission in the control of apoptosis

1 reference

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21 January 2018

The mechanism of mitochondrial membrane potential retention following release of cytochrome c in apoptotic GT1-7 neural cells

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21 January 2018

Functional F1-ATPase essential in maintaining growth and membrane potential of human mitochondrial DNA-depleted rho degrees cells

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21 January 2018

Mitochondrial membrane potential and neuronal glutamate excitotoxicity: mortality and millivolts

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Different responses of astrocytes and neurons to nitric oxide: the role of glycolytically generated ATP in astrocyte protection.

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DeltaPsi(m)-Dependent and -independent production of reactive oxygen species by rat brain mitochondria

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Mitochondrial superoxide: production, biological effects, and activation of uncoupling proteins

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Superoxide production by NADH:ubiquinone oxidoreductase (complex I) depends on the pH gradient across the mitochondrial inner membrane

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Triggering and modulation of apoptosis by oxidative stress

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Granzyme A induces caspase-independent mitochondrial damage, a required first step for apoptosis

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The role of oxidative stress in carcinogenesis

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Disruption of mitochondrial function during apoptosis is mediated by caspase cleavage of the p75 subunit of complex I of the electron transport chain

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NADH oxidase activity of mitochondrial apoptosis-inducing factor

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A systematic RNAi screen identifies a critical role for mitochondria in C. elegans longevity

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Identifiers

10.1093/CARCIN/BGI315

1 reference

Europe PubMed Central

25 December 2017

http://europepmc.org/abstract/MED/16361271

1 reference

Europe PubMed Central

25 December 2017

http://europepmc.org/abstract/MED/16361271

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