(Q48311934)

10 February 2018

title

Effect of chronic lead exposure on pro-apoptotic Bax and anti-apoptotic Bcl-2 protein expression in rat hippocampus in vivo. (English)

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10 February 2018

ORCID Public Data File 2021

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author name string

Ali M Sharifi

series ordinal

1

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10 February 2018

Seyed Hadi Mousavi

series ordinal

2

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10 February 2018

Masoumeh Jorjani

series ordinal

3

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10 February 2018

publication date

11 February 2010

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10 February 2018

Cellular and Molecular Neurobiology

published in

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10 February 2018

volume

30

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10 February 2018

issue

5

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10 February 2018

page(s)

769-774

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10 February 2018

https://scigraph.springernature.com/pub.10.1007/s10571-010-9504-1

exact match

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cites work

Blood lead, hearing thresholds, and neurobehavioral development in children and youth.

1 reference

Crossref

https://api.crossref.org/works/10.1007%2FS10571-010-9504-1

Bad, a heterodimeric partner for Bcl-XL and Bcl-2, displaces Bax and promotes cell death

21 January 2018

1 reference

12 December 2020

bcl-x, a bcl-2-related gene that functions as a dominant regulator of apoptotic cell death

1 reference

12 December 2020

Evidence that lead acts as a calcium substitute in second messenger metabolism.

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12 December 2020

Early lead exposure and the hippocampus: a review and recent advances

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12 December 2020

Neurobehavioral aspects of lead neurotoxicity in children.

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12 December 2020

[Effects of lead on the Brn-3a expression and the apoptosis in hippocampus neurons]

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12 December 2020

Investigation of acute lead poisoning on apoptosis in rat hippocampus in vivo.

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12 December 2020

Molecular thanatopsis: a discourse on the BCL2 family and cell death.

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12 December 2020

Increase in bax expression in substantia nigra following 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine (MPTP) treatment of mice.

1 reference

12 December 2020

Effect of lead acetate on the apoptosis and the expression of bcl-2 and bax genes in rat brain cells

1 reference

12 December 2020

Characterization of A1, a novel hemopoietic-specific early-response gene with sequence similarity to bcl-2

1 reference

12 December 2020

MCL1, a gene expressed in programmed myeloid cell differentiation, has sequence similarity to BCL2

1 reference

12 December 2020

Suppression of programmed neuronal death by sustained elevation of cytoplasmic calcium

1 reference

12 December 2020

Low-level lead exposure, intelligence and academic achievement: a long-term follow-up study

1 reference

12 December 2020

Induction of apoptosis by the Bcl-2 homologue Bak

1 reference

12 December 2020

Bcl-xL overexpression blocks bax-mediated mitochondrial contact site formation and apoptosis in rod photoreceptors of lead-exposed mice

1 reference

12 December 2020

Cloning and functional analysis of BAG-1: a novel Bcl-2-binding protein with anti-cell death activity

1 reference

12 December 2020

Lead and calcium produce rod photoreceptor cell apoptosis by opening the mitochondrial permeability transition pore.

1 reference

12 December 2020

Proapoptotic protein Bax heterodimerizes with Bcl-2 and homodimerizes with Bax via a novel domain (BH3) distinct from BH1 and BH2

1 reference

12 December 2020

Protective effects of ascorbic acid against lead-induced apoptotic neurodegeneration in the developing rat hippocampus in vivo

1 reference

12 December 2020

Impaired neuropsychological functioning in lead-exposed children.

1 reference

12 December 2020

Studying the effects of lead on DNA fragmentation and proapoptotic bax and antiapoptotic bcl-2 protein expression in PC12 cells

1 reference

12 December 2020

The effects of meso-2,3-dimercaptosuccinic acid and oligomeric procyanidins on acute lead neurotoxicity in rat hippocampus

1 reference

12 December 2020

Neuropsychological studies in children with elevated tooth-lead concentrations. II. Extended study.

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12 December 2020

Glutamate neurotoxicity and diseases of the nervous system

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12 December 2020

The relationship between blood-lead concentrations, intelligence, attainment and behaviour in a school population: the second London study

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12 December 2020

Postnatal inorganic lead exposure decreases the number of spontaneously active midbrain dopamine neurons in the rat.

1 reference

12 December 2020

Bcl-2 is an inner mitochondrial membrane protein that blocks programmed cell death

1 reference

12 December 2020

Lead-induced alteration of apoptotic proteins in different regions of adult rat brain

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12 December 2020

Low-level lead exposure and children's cognitive function in the preschool years

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12 December 2020

Molecular targets of lead in brain neurotoxicity

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12 December 2020

Bik, a novel death-inducing protein shares a distinct sequence motif with Bcl-2 family proteins and interacts with viral and cellular survival-promoting proteins

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12 December 2020

Lead-induced alterations of apoptosis and neurotrophic factor mRNA in the developing rat cortex, hippocampus, and cerebellum

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12 December 2020

Bax suppresses tumorigenesis and stimulates apoptosis in vivo

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12 December 2020

Bcl-2 family proteins and the hormonal control of cell life and death in normalcy and neoplasia.

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12 December 2020

Differential effects of lead exposure on components of verbal memory

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12 December 2020

Lead neurotoxicity in children: basic mechanisms and clinical correlates

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12 December 2020

Bcl-2 heterodimerizes in vivo with a conserved homolog, Bax, that accelerates programmed cell death

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12 December 2020

BAX-induced cell death may not require interleukin 1 beta-converting enzyme-like proteases

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12 December 2020

Brain capillaries: a target for inorganic lead poisoning

1 reference

12 December 2020

Lead (Pb+2) promotes apoptosis in newborn rat cerebellar neurons: pathological implications.

1 reference

12 December 2020

10.1007/S10571-010-9504-1

Identifiers

DOI

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10 February 2018

PubMed ID

20148304

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10 February 2018