(Q48635943)

13 February 2018

title

Immunization reverses memory deficits without reducing brain Abeta burden in Alzheimer's disease model. (English)

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13 February 2018

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David M Holtzman

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13 February 2018

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Stephen J Greene

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13 February 2018

author name string

Jean-Cosme Dodart

1

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13 February 2018

Kelly R Bales

2

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Kimberley S Gannon

3

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13 February 2018

Ronald B DeMattos

5

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Chantal Mathis

6

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Cynthia A DeLong

7

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13 February 2018

Su Wu

8

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13 February 2018

Xin Wu

9

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13 February 2018

Steven M Paul

11

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language of work or name

English

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publication date

1 May 2002

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13 February 2018

published in

Nature Neuroscience

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13 February 2018

volume

5

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13 February 2018

page(s)

452-457

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13 February 2018

issue

5

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13 February 2018

https://scigraph.springernature.com/pub.10.1038/nn842

exact match

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cites work

Alzheimer's disease: the amyloid cascade hypothesis

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7 January 2021

Cell biology of the amyloid beta-protein precursor and the mechanism of Alzheimer's disease

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7 January 2021

Alzheimer's disease: a re-examination of the amyloid hypothesis

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7 January 2021

Immunization with amyloid-beta attenuates Alzheimer-disease-like pathology in the PDAPP mouse

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7 January 2021

Peripherally administered antibodies against amyloid beta-peptide enter the central nervous system and reduce pathology in a mouse model of Alzheimer disease

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7 January 2021

A beta peptide immunization reduces behavioural impairment and plaques in a model of Alzheimer's disease

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7 January 2021

A beta peptide vaccination prevents memory loss in an animal model of Alzheimer's disease.

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7 January 2021

Peripheral anti-A beta antibody alters CNS and plasma A beta clearance and decreases brain A beta burden in a mouse model of Alzheimer's disease

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7 January 2021

Immunization with a nontoxic/nonfibrillar amyloid-beta homologous peptide reduces Alzheimer's disease-associated pathology in transgenic mice

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7 January 2021

Behavioral disturbances in transgenic mice overexpressing the V717F beta-amyloid precursor protein.

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7 January 2021

Behavioral deficits in APP(V717F) transgenic mice deficient for the apolipoprotein E gene.

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7 January 2021

Neuroanatomical abnormalities in behaviorally characterized APP(V717F) transgenic mice.

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7 January 2021

The beta-amyloid precursor protein and its derivatives: from biology to learning and memory processes

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7 January 2021

Soluble amyloid beta peptide concentration as a predictor of synaptic change in Alzheimer's disease

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7 January 2021

Soluble pool of Abeta amyloid as a determinant of severity of neurodegeneration in Alzheimer's disease

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7 January 2021

Specific spatial learning deficits become severe with age in beta -amyloid precursor protein transgenic mice that harbor diffuse beta -amyloid deposits but do not form plaques

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7 January 2021

Alzheimer-type neuropathology in transgenic mice overexpressing V717F β-amyloid precursor protein

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7 January 2021

Isolation and quantification of soluble Alzheimer's beta-peptide from biological fluids

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7 January 2021

A new one-trial test for neurobiological studies of memory in rats. 1: Behavioral data

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7 January 2021

Scopolamine-induced deficits in a two-trial object recognition task in mice.

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7 January 2021

Apolipoprotein E is essential for amyloid deposition in the APP(V717F) transgenic mouse model of Alzheimer's disease

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7 January 2021

Amyloid precursor protein processing and A beta42 deposition in a transgenic mouse model of Alzheimer disease

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7 January 2021

Dimensions Publication ID

Identifiers

DOI

10.1038/NN842

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13 February 2018