(Q50336551)

English

Fas-associated death domain protein (FADD) and caspase-8 mediate up-regulation of c-Fos by Fas ligand and tumor necrosis factor-related apoptosis-inducing ligand (TRAIL) via a FLICE inhibitory protein (FLIP)-regulated pathway.

scientific article published on 30 May 2001

In more languages

Statements

scholarly article

1 reference

Europe PubMed Central

PubMed publication ID

5 March 2018

http://europepmc.org/abstract/MED/11384965

Fas-associated death domain protein (FADD) and caspase-8 mediate up-regulation of c-Fos by Fas ligand and tumor necrosis factor-related apoptosis-inducing ligand (TRAIL) via a FLICE inhibitory protein (FLIP)-regulated pathway. (English)

1 reference

Europe PubMed Central

PubMed publication ID

5 March 2018

http://europepmc.org/abstract/MED/11384965

10

1 reference

Europe PubMed Central

PubMed publication ID

5 March 2018

http://europepmc.org/abstract/MED/11384965

9

object named as

Tschopp J

1 reference

Europe PubMed Central

PubMed publication ID

5 March 2018

Peter Scheurich

8

object named as

Scheurich P

1 reference

Europe PubMed Central

PubMed publication ID

5 March 2018

http://europepmc.org/abstract/MED/11384965

author name string

Siegmund D

1

1 reference

Europe PubMed Central

PubMed publication ID

5 March 2018

http://europepmc.org/abstract/MED/11384965

Mauri D

2

1 reference

Europe PubMed Central

PubMed publication ID

5 March 2018

http://europepmc.org/abstract/MED/11384965

Peters N

3

1 reference

Europe PubMed Central

PubMed publication ID

5 March 2018

http://europepmc.org/abstract/MED/11384965

Juo P

4

1 reference

Europe PubMed Central

PubMed publication ID

5 March 2018

http://europepmc.org/abstract/MED/11384965

Thome M

5

1 reference

Europe PubMed Central

PubMed publication ID

5 March 2018

http://europepmc.org/abstract/MED/11384965

Reichwein M

6

1 reference

Europe PubMed Central

PubMed publication ID

5 March 2018

http://europepmc.org/abstract/MED/11384965

Blenis J

7

1 reference

Europe PubMed Central

PubMed publication ID

5 March 2018

http://europepmc.org/abstract/MED/11384965

language of work or name

0 references

publication date

30 May 2001

1 reference

Europe PubMed Central

PubMed publication ID

5 March 2018

http://europepmc.org/abstract/MED/11384965

Journal of Biological Chemistry

1 reference

Europe PubMed Central

PubMed publication ID

5 March 2018

http://europepmc.org/abstract/MED/11384965

276

1 reference

Europe PubMed Central

PubMed publication ID

5 March 2018

http://europepmc.org/abstract/MED/11384965

35

1 reference

Europe PubMed Central

PubMed publication ID

5 March 2018

http://europepmc.org/abstract/MED/11384965

32585-32590

1 reference

Europe PubMed Central

PubMed publication ID

5 March 2018

http://europepmc.org/abstract/MED/11384965

Fas and perforin pathways as major mechanisms of T cell-mediated cytotoxicity

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M100444200

21 January 2018

Fas ligand mediates activation-induced cell death in human T lymphocytes

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M100444200

21 January 2018

Cell-autonomous Fas (CD95)/Fas-ligand interaction mediates activation-induced apoptosis in T-cell hybridomas.

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M100444200

21 January 2018

Autocrine T-cell suicide mediated by APO-1/(Fas/CD95)

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M100444200

21 January 2018

Fas(CD95)/FasL interactions required for programmed cell death after T-cell activation.

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M100444200

21 January 2018

Lymphoproliferation disorder in mice explained by defects in Fas antigen that mediates apoptosis

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M100444200

21 January 2018

Generalized lymphoproliferative disease in mice, caused by a point mutation in the Fas ligand

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M100444200

21 January 2018

Mutations in Fas associated with human lymphoproliferative syndrome and autoimmunity

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M100444200

21 January 2018

Fas ligand mutation in a patient with systemic lupus erythematosus and lymphoproliferative disease

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M100444200

21 January 2018

CD95's deadly mission in the immune system

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M100444200

21 January 2018

The biochemistry of apoptosis

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M100444200

21 January 2018

Viral FLICE-inhibitory proteins (FLIPs) prevent apoptosis induced by death receptors

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M100444200

21 January 2018

Casper is a FADD- and caspase-related inducer of apoptosis

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M100444200

21 January 2018

CLARP, a death effector domain-containing protein interacts with caspase-8 and regulates apoptosis

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M100444200

21 January 2018

FLAME-1, a novel FADD-like anti-apoptotic molecule that regulates Fas/TNFR1-induced apoptosis

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M100444200

21 January 2018

I-FLICE, a novel inhibitor of tumor necrosis factor receptor-1- and CD-95-induced apoptosis

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M100444200

21 January 2018

CASH, a novel caspase homologue with death effector domains

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M100444200

21 January 2018

MRIT, a novel death-effector domain-containing protein, interacts with caspases and BclXL and initiates cell death

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M100444200

21 January 2018

Cell death attenuation by 'Usurpin', a mammalian DED-caspase homologue that precludes caspase-8 recruitment and activation by the CD-95 (Fas, APO-1) receptor complex

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M100444200

21 January 2018

The role of c-FLIP in modulation of CD95-induced apoptosis

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M100444200

21 January 2018

The CD95 (APO-1/Fas) receptor activates NF-kappaB independently of its cytotoxic function

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M100444200

21 January 2018

Inhibition of death receptor-mediated gene induction by a cycloheximide-sensitive factor occurs at the level of or upstream of Fas-associated death domain protein (FADD)

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M100444200

21 January 2018

The death domain kinase RIP is essential for TRAIL (Apo2L)-induced activation of IkappaB kinase and c-Jun N-terminal kinase

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M100444200

21 January 2018

Fas signal transduction triggers either proliferation or apoptosis in human fibroblasts

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M100444200

21 January 2018

Fas engagement induces the maturation of dendritic cells (DCs), the release of interleukin (IL)-1beta, and the production of interferon gamma in the absence of IL-12 during DC-T cell cognate interaction: a new role for Fas ligand in inflammatory res

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M100444200

21 January 2018

Essential requirement for caspase-8/FLICE in the initiation of the Fas-induced apoptotic cascade

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M100444200

21 January 2018

Fas-mediated apoptosis and activation-induced T-cell proliferation are defective in mice lacking FADD/Mort1

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M100444200

21 January 2018

A dominant interfering mutant of FADD/MORT1 enhances deletion of autoreactive thymocytes and inhibits proliferation of mature T lymphocytes

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M100444200

21 January 2018

A role for FADD in T cell activation and development

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M100444200

21 January 2018

Surface expression of TRAIL/Apo-2 ligand in activated mouse T and B cells

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M100444200

21 January 2018

Regulation of the c-fos promoter

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M100444200

21 January 2018

Caspase-independent cell killing by Fas-associated protein with death domain

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M100444200

21 January 2018

Activation of NF-kappaB by FADD, Casper, and caspase-8.

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M100444200

21 January 2018

Activation of the c-Jun N-terminal kinase/stress-activated protein kinase pathway by overexpression of caspase-8 and its homologs

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M100444200

21 January 2018

TRAIL receptor-2 signals apoptosis through FADD and caspase-8

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M100444200

21 January 2018

FADD/MORT1 and caspase-8 are recruited to TRAIL receptors 1 and 2 and are essential for apoptosis mediated by TRAIL receptor 2

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M100444200

21 January 2018

Apo2L/TRAIL-dependent recruitment of endogenous FADD and caspase-8 to death receptors 4 and 5.

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M100444200

21 January 2018

FADD is required for DR4- and DR5-mediated apoptosis: lack of trail-induced apoptosis in FADD-deficient mouse embryonic fibroblasts

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M100444200

21 January 2018

TRAIL/Apo2L activates c-Jun NH2-terminal kinase (JNK) via caspase-dependent and caspase-independent pathways

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M100444200

21 January 2018

The caspase-8 inhibitor FLIP promotes activation of NF-kappaB and Erk signaling pathways

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M100444200

21 January 2018

Fas transduces activation signals in normal human T lymphocytes

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M100444200

21 January 2018

Caspase activation is required for T cell proliferation

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M100444200

21 January 2018

Dichotomy between naïve and memory CD4(+) T cell responses to Fas engagement

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M100444200

21 January 2018

Early activation of caspases during T lymphocyte stimulation results in selective substrate cleavage in nonapoptotic cells

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M100444200

21 January 2018

Bone and haematopoietic defects in mice lacking c-fos

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M100444200

21 January 2018

Cytolytic T-cell cytotoxicity is mediated through perforin and Fas lytic pathways

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M100444200

21 January 2018

A role for CD95 ligand in preventing graft rejection

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M100444200

21 January 2018

Identifiers

10.1074/JBC.M100444200

1 reference

Europe PubMed Central

PubMed publication ID

5 March 2018

http://europepmc.org/abstract/MED/11384965

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

5 March 2018

http://europepmc.org/abstract/MED/11384965

Sitelinks

Wikipedia(0 entries)

Wikibooks(0 entries)

Wikinews(0 entries)

Wikiquote(0 entries)

Wikisource(0 entries)

Wikiversity(0 entries)

Wikivoyage(0 entries)

Wiktionary(0 entries)

Multilingual sites(0 entries)

Retrieved from "https://www.wikidata.org/w/index.php?title=Q50336551&oldid=2118678561"