(Q53377245)

13 May 2018

title

Both products of the mouse Ink4a/Arf locus suppress melanoma formation in vivo. (English)

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13 May 2018

author

Norman Sharpless

1

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13 May 2018

Lynda Chin

5

object named as

Lynda Chin

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13 May 2018

author name string

Karuppiah Kannan

2

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13 May 2018

Jin Xu

3

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13 May 2018

Marcus Wolfram Bosenberg

4

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13 May 2018

language of work or name

English

0 references

publication date

1 August 2003

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13 May 2018

published in

Oncogene

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13 May 2018

volume

22

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13 May 2018

issue

32

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13 May 2018

page(s)

5055-5059

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13 May 2018

Oncogenic ras activates the ARF-p53 pathway to suppress epithelial cell transformation

cites work

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1206809

Invasive melanoma in Cdk4-targeted mice

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1206809

Mutations of the BRAF gene in human cancer

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1206809

7 January 2021

Tumor suppression at the mouse INK4a locus mediated by the alternative reading frame product p19ARF

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1206809

7 January 2021

Loss of p16Ink4a confers susceptibility to metastatic melanoma in mice

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1206809

7 January 2021

Distinct roles of nerve and muscle in postsynaptic differentiation of the neuromuscular synapse

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1206809

7 January 2021

p19ARF links the tumour suppressor p53 to Ras

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1206809

7 January 2021

Hypermethylation of the p16 (Ink4a) promoter in B6C3F1 mouse primary lung adenocarcinomas and mouse lung cell lines

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1206809

7 January 2021

A melanoma-associated germline mutation in exon 1beta inactivates p14ARF.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1206809

7 January 2021

Homozygous deletions of methylthioadenosine phosphorylase (MTAP) are more frequent than p16INK4A (CDKN2) homozygous deletions in primary non-small cell lung cancers (NSCLC).

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1206809

7 January 2021

Role of the INK4a locus in tumor suppression and cell mortality.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1206809

7 January 2021

Oncogenic ras provokes premature cell senescence associated with accumulation of p53 and p16INK4a

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1206809

7 January 2021

Loss of p16Ink4a with retention of p19Arf predisposes mice to tumorigenesis

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1206809

7 January 2021

Identifiers

DOI

10.1038/SJ.ONC.1206809

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13 May 2018

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13 May 2018