(Q64982207)

2 July 2019

title

Tau impairs neural circuits, dominating amyloid-β effects, in Alzheimer models in vivo. (English)

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2 July 2019

author

Marc Aurel Busche

1

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2 July 2019

Simon Dujardin

3

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2 July 2019

Israel Nelken

9

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2 July 2019

Bradley Hyman

10

object named as

Bradley T Hyman

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2 July 2019

Susanne Wegmann

2

1 reference

2 July 2019

Caitlin Commins

4

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2 July 2019

Julia Schiantarelli

5

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2 July 2019

Naomi Klickstein

6

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2 July 2019

Tarun V Kamath

7

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2 July 2019

George A Carlson

8

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2 July 2019

publication date

17 December 2018

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2 July 2019

published in

Nature Neuroscience

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2 July 2019

volume

22

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2 July 2019

issue

1

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2 July 2019

page(s)

57-64

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2 July 2019

describes a project that uses

ImageJ

1 reference

11 June 2022

https://www.ebi.ac.uk/europepmc/webservices/rest/PMC6560629/fullTextXML

inferred from PubMed Central ID database lookup

exact match

https://scigraph.springernature.com/pub.10.1038/s41593-018-0289-8

0 references

cites work

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7 January 2021

Neuropathological stageing of Alzheimer-related changes

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7 January 2021

The Topographical and Neuroanatomical Distribution of Neurofibrillary Tangles and Neuritic Plaques in the Cerebral Cortex of Patients with Alzheimer's Disease

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7 January 2021

PET Imaging of Tau Deposition in the Aging Human Brain.

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7 January 2021

The biochemical pathway of neurofibrillary degeneration in aging and Alzheimer's disease.

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https://api.crossref.org/works/10.1038%2FS41593-018-0289-8

7 January 2021

Evaluation of Tau Imaging in Staging Alzheimer Disease and Revealing Interactions Between β-Amyloid and Tauopathy

1 reference

https://api.crossref.org/works/10.1038%2FS41593-018-0289-8

7 January 2021

Enhanced neurofibrillary degeneration in transgenic mice expressing mutant tau and APP.

1 reference

https://api.crossref.org/works/10.1038%2FS41593-018-0289-8

7 January 2021

Formation of neurofibrillary tangles in P301l tau transgenic mice induced by Abeta 42 fibrils

1 reference

https://api.crossref.org/works/10.1038%2FS41593-018-0289-8

7 January 2021

A{beta} accelerates the spatiotemporal progression of tau pathology and augments tau amyloidosis in an Alzheimer mouse model

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7 January 2021

Enhanced Tau Aggregation in the Presence of Amyloid β.

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7 January 2021

Structural tract alterations predict downstream tau accumulation in amyloid-positive older individuals.

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7 January 2021

Association Between Amyloid and Tau Accumulation in Young Adults With Autosomal Dominant Alzheimer Disease.

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7 January 2021

Amyloid-β plaques enhance Alzheimer's brain tau-seeded pathologies by facilitating neuritic plaque tau aggregation.

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https://api.crossref.org/works/10.1038%2FS41593-018-0289-8

7 January 2021

Imaging input and output of neocortical networks in vivo

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https://api.crossref.org/works/10.1038%2FS41593-018-0289-8

7 January 2021

Ultrasensitive fluorescent proteins for imaging neuronal activity

1 reference

https://api.crossref.org/works/10.1038%2FS41593-018-0289-8

7 January 2021

Clusters of hyperactive neurons near amyloid plaques in a mouse model of Alzheimer's disease.

1 reference

https://api.crossref.org/works/10.1038%2FS41593-018-0289-8

7 January 2021

Staged decline of neuronal function in vivo in an animal model of Alzheimer's disease

1 reference

https://api.crossref.org/works/10.1038%2FS41593-018-0289-8

7 January 2021

Decreased amyloid-β and increased neuronal hyperactivity by immunotherapy in Alzheimer's models

1 reference

https://api.crossref.org/works/10.1038%2FS41593-018-0289-8

7 January 2021

BACE inhibition-dependent repair of Alzheimer's pathophysiology

1 reference

https://api.crossref.org/works/10.1038%2FS41593-018-0289-8

7 January 2021

Human tau increases amyloid β plaque size but not amyloid β-mediated synapse loss in a novel mouse model of Alzheimer's disease

1 reference

https://api.crossref.org/works/10.1038%2FS41593-018-0289-8

7 January 2021

Tau suppression in a neurodegenerative mouse model improves memory function

1 reference

https://api.crossref.org/works/10.1038%2FS41593-018-0289-8

7 January 2021

Accumulation of pathological tau species and memory loss in a conditional model of tauopathy.

1 reference

https://api.crossref.org/works/10.1038%2FS41593-018-0289-8

7 January 2021

Caspase activation precedes and leads to tangles

1 reference

https://api.crossref.org/works/10.1038%2FS41593-018-0289-8

7 January 2021

Tau and amyloid-related pathologies in the entorhinal cortex have divergent effects in the hippocampal circuit.

1 reference

https://api.crossref.org/works/10.1038%2FS41593-018-0289-8

7 January 2021

Reducing endogenous tau ameliorates amyloid beta-induced deficits in an Alzheimer's disease mouse model

1 reference

https://api.crossref.org/works/10.1038%2FS41593-018-0289-8

7 January 2021

Dendritic function of tau mediates amyloid-beta toxicity in Alzheimer's disease mouse models

1 reference

https://api.crossref.org/works/10.1038%2FS41593-018-0289-8

7 January 2021

Antisense reduction of tau in adult mice protects against seizures

1 reference

https://api.crossref.org/works/10.1038%2FS41593-018-0289-8

7 January 2021

What Happens with the Circuit in Alzheimer's Disease in Mice and Humans?

1 reference

https://api.crossref.org/works/10.1038%2FS41593-018-0289-8

7 January 2021

Positron emission tomography in evaluation of dementia: Regional brain metabolism and long-term outcome

1 reference

https://api.crossref.org/works/10.1038%2FS41593-018-0289-8

7 January 2021

Longitudinal PET Evaluation of Cerebral Metabolic Decline in Dementia: A Potential Outcome Measure in Alzheimer's Disease Treatment Studies

1 reference

https://api.crossref.org/works/10.1038%2FS41593-018-0289-8

7 January 2021

Default-mode network activity distinguishes Alzheimer's disease from healthy aging: evidence from functional MRI

1 reference

https://api.crossref.org/works/10.1038%2FS41593-018-0289-8

7 January 2021

Cerebral perfusion SPET correlated with Braak pathological stage in Alzheimer's disease.

1 reference

https://api.crossref.org/works/10.1038%2FS41593-018-0289-8

7 January 2021

Apolipoprotein E epsilon4 allele decreases functional connectivity in Alzheimer's disease as measured by EEG coherence

1 reference

https://api.crossref.org/works/10.1038%2FS41593-018-0289-8

7 January 2021

Neurofibrillary tangles but not senile plaques parallel duration and severity of Alzheimer's disease

1 reference

https://api.crossref.org/works/10.1038%2FS41593-018-0289-8

7 January 2021

Correlation of Alzheimer disease neuropathologic changes with cognitive status: a review of the literature

1 reference

https://api.crossref.org/works/10.1038%2FS41593-018-0289-8

7 January 2021

Network abnormalities and interneuron dysfunction in Alzheimer disease

1 reference

https://api.crossref.org/works/10.1038%2FS41593-018-0289-8

7 January 2021

Increased hippocampal activation in mild cognitive impairment compared to normal aging and AD

1 reference

https://api.crossref.org/works/10.1038%2FS41593-018-0289-8

7 January 2021

Reduction of hippocampal hyperactivity improves cognition in amnestic mild cognitive impairment

1 reference

https://api.crossref.org/works/10.1038%2FS41593-018-0289-8

7 January 2021

Molecular drivers and cortical spread of lateral entorhinal cortex dysfunction in preclinical Alzheimer's disease

1 reference

https://api.crossref.org/works/10.1038%2FS41593-018-0289-8

7 January 2021

Tau mislocalization to dendritic spines mediates synaptic dysfunction independently of neurodegeneration

1 reference

https://api.crossref.org/works/10.1038%2FS41593-018-0289-8

7 January 2021

Pathological tau disrupts ongoing network activity.

1 reference

https://api.crossref.org/works/10.1038%2FS41593-018-0289-8

7 January 2021

Hyperphosphorylated tau causes reduced hippocampal CA1 excitability by relocating the axon initial segment

1 reference

https://api.crossref.org/works/10.1038%2FS41593-018-0289-8

7 January 2021

Reduction of soluble Abeta and tau, but not soluble Abeta alone, ameliorates cognitive decline in transgenic mice with plaques and tangles

1 reference

https://api.crossref.org/works/10.1038%2FS41593-018-0289-8

7 January 2021

Tau-induced defects in synaptic plasticity, learning, and memory are reversible in transgenic mice after switching off the toxic Tau mutant.

1 reference

https://api.crossref.org/works/10.1038%2FS41593-018-0289-8

7 January 2021

Tau oligomers impair memory and induce synaptic and mitochondrial dysfunction in wild-type mice.

1 reference

https://api.crossref.org/works/10.1038%2FS41593-018-0289-8

7 January 2021

Cognitive defects are reversible in inducible mice expressing pro-aggregant full-length human Tau.

1 reference

https://api.crossref.org/works/10.1038%2FS41593-018-0289-8

7 January 2021

Passive immunization with Tau oligomer monoclonal antibody reverses tauopathy phenotypes without affecting hyperphosphorylated neurofibrillary tangles.

1 reference

https://api.crossref.org/works/10.1038%2FS41593-018-0289-8

7 January 2021

Tau pathology does not affect experience-driven single-neuron and network-wide Arc/Arg3.1 responses

1 reference

https://api.crossref.org/works/10.1038%2FS41593-018-0289-8

7 January 2021

Neurofibrillary tangle-bearing neurons are functionally integrated in cortical circuits in vivo

1 reference

https://api.crossref.org/works/10.1038%2FS41593-018-0289-8

7 January 2021

Mutant presenilins specifically elevate the levels of the 42 residue beta-amyloid peptide in vivo: evidence for augmentation of a 42-specific gamma secretase

1 reference

https://api.crossref.org/works/10.1038%2FS41593-018-0289-8

7 January 2021

Control of memory formation through regulated expression of a CaMKII transgene

1 reference

https://api.crossref.org/works/10.1038%2FS41593-018-0289-8

7 January 2021

Population code in mouse V1 facilitates readout of natural scenes through increased sparseness

1 reference

https://api.crossref.org/works/10.1038%2FS41593-018-0289-8

7 January 2021

Relationships between flortaucipir PET tau binding and amyloid burden, clinical diagnosis, age and cognition

1 reference

PubMed

https://pubmed.ncbi.nlm.nih.gov/30559471

12 December 2020

inferred from PubMed ID database lookup

Identifiers

DOI

10.1038/S41593-018-0289-8

1 reference

2 July 2019

Dimensions Publication ID

0 references

1 reference

2 July 2019

PubMed ID

30559471

1 reference

2 July 2019