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Defective embryonic neurogenesis in Ku-deficient but not DNA-dependent protein kinase catalytic subunit-deficient mice
scientific journal article
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instance of
scholarly article
1 reference
stated in
PubMed
PubMed ID
10716994
retrieved
24 January 2017
title
Defective embryonic neurogenesis in Ku-deficient but not DNA-dependent protein kinase catalytic subunit-deficient mice
(English)
1 reference
stated in
PubMed
PubMed ID
10716994
retrieved
24 January 2017
main subject
Ligase IV, DNA, ATP-dependent
1 reference
stated in
GOA release 2020-03-11
X-ray repair complementing defective repair in Chinese hamster cells 5
1 reference
stated in
GOA release 2020-03-11
X-ray repair complementing defective repair in Chinese hamster cells 6
1 reference
stated in
GOA release 2020-03-11
X-ray repair complementing defective repair in Chinese hamster cells 4
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stated in
GOA release 2020-03-11
neurogenesis
1 reference
based on heuristic
inferred from title
author name string
Y. Gu
series ordinal
1
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stated in
PubMed
PubMed ID
10716994
retrieved
24 January 2017
J. Sekiguchi
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2
1 reference
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PubMed
PubMed ID
10716994
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24 January 2017
Y. Gao
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3
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PubMed
PubMed ID
10716994
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24 January 2017
P. Dikkes
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4
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PubMed
PubMed ID
10716994
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24 January 2017
K. Frank
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5
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PubMed
PubMed ID
10716994
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24 January 2017
D. Ferguson
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6
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PubMed
PubMed ID
10716994
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24 January 2017
P. Hasty
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7
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PubMed
PubMed ID
10716994
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24 January 2017
J. Chun
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8
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PubMed
PubMed ID
10716994
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24 January 2017
F. W. Alt
series ordinal
9
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PubMed
PubMed ID
10716994
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24 January 2017
language of work or name
English
0 references
publication date
14 March 2000
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PubMed
PubMed ID
10716994
retrieved
24 January 2017
published in
Proceedings of the National Academy of Sciences of the United States of America
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PubMed
PubMed ID
10716994
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24 January 2017
volume
97
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PubMed
PubMed ID
10716994
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24 January 2017
page(s)
2668–2673
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PubMed
PubMed ID
10716994
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24 January 2017
issue
6
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PubMed
PubMed ID
10716994
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24 January 2017
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Targeted disruption of the gene encoding DNA ligase IV leads to lethality in embryonic mice
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A critical role for DNA end-joining proteins in both lymphogenesis and neurogenesis
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19 March 2017
Late embryonic lethality and impaired V(D)J recombination in mice lacking DNA ligase IV
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DNA ligase IV is essential for V(D)J recombination and DNA double-strand break repair in human precursor lymphocytes
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Targeted disruption of the catalytic subunit of the DNA-PK gene in mice confers severe combined immunodeficiency and radiosensitivity
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Diversity revealed by a novel family of cadherins expressed in neurons at a synaptic complex
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Ku70-deficient embryonic stem cells have increased ionizing radiosensitivity, defective DNA end-binding activity, and inability to support V(D)J recombination
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RAG-1-deficient mice have no mature B and T lymphocytes
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RAG-2-deficient mice lack mature lymphocytes owing to inability to initiate V(D)J rearrangement
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Rearranging views on neurogenesis: neuronal death in the absence of DNA end-joining proteins
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Catalytic subunit of DNA-dependent protein kinase: impact on lymphocyte development and tumorigenesis
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Identification of genes involved in repair of DNA double-strand breaks in mammalian cells.
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Hypersensitivity of Ku80-deficient cell lines and mice to DNA damage: the effects of ionizing radiation on growth, survival, and development
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Ku70 is required for DNA repair but not for T cell antigen receptor gene recombination In vivo
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DNA double-strand breaks caused by replication arrest
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Developmental regulation of V(D)J recombination and lymphocyte differentiation
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Ku86-deficient mice exhibit severe combined immunodeficiency and defective processing of V(D)J recombination intermediates
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Requirement for Ku80 in growth and immunoglobulin V(D)J recombination
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Programmed cell death is a universal feature of embryonic and postnatal neuroproliferative regions throughout the central nervous system.
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Crossref
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21 January 2018
A targeted DNA-PKcs-null mutation reveals DNA-PK-independent functions for KU in V(D)J recombination
1 reference
stated in
PubMed
reference URL
https://pubmed.ncbi.nlm.nih.gov/10716994
retrieved
12 December 2020
based on heuristic
inferred from PubMed ID database lookup
Repair of ionizing radiation damage in mammalian cells. Alternative pathways and their fidelity
1 reference
stated in
PubMed
reference URL
https://pubmed.ncbi.nlm.nih.gov/10716994
retrieved
12 December 2020
based on heuristic
inferred from PubMed ID database lookup
Identifiers
DOI
10.1073/PNAS.97.6.2668
0 references
ADS bibcode
2000PNAS...97.2668G
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PMCID
15987
0 references
PubMed ID
10716994
1 reference
stated in
PubMed
PubMed ID
10716994
retrieved
24 January 2017
ResearchGate publication ID
12598871
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