(Q34247072)

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ARK5 suppresses the cell death induced by nutrient starvation and death receptors via inhibition of caspase 8 activation, but not by chemotherapeutic agents or UV irradiation

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scholarly article

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Europe PubMed Central

31 July 2017

ARK5 suppresses the cell death induced by nutrient starvation and death receptors via inhibition of caspase 8 activation, but not by chemotherapeutic agents or UV irradiation (English)

1 reference

Europe PubMed Central

31 July 2017

programmed necrotic cell death in response to starvation

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Gene Ontology release 2020-05-02

Gene Ontology ID

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based on heuristic

inferred from title

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based on heuristic

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6

object named as

Hiroyasu Esumi

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Europe PubMed Central

31 July 2017

5

object named as

Tsutomu Ogura

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Europe PubMed Central

31 July 2017

author name string

Atsushi Suzuki

1

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Europe PubMed Central

31 July 2017

Gen-Ichi Kusakai

2

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Europe PubMed Central

31 July 2017

Atsuhiro Kishimoto

3

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Europe PubMed Central

31 July 2017

Jie Lu

4

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Europe PubMed Central

31 July 2017

language of work or name

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publication date

1 September 2003

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Europe PubMed Central

31 July 2017

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Europe PubMed Central

31 July 2017

22

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31 July 2017

6177-6182

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Europe PubMed Central

31 July 2017

40

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31 July 2017

https://scigraph.springernature.com/pub.10.1038/sj.onc.1206899

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Human ICE/CED-3 protease nomenclature

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Phosphoinositide-3-OH kinase-dependent regulation of glycogen synthase kinase 3 and protein kinase B/AKT by the integrin-linked kinase

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Hypoxia and nitric oxide treatment confer tolerance to glucose starvation in a 5'-AMP-activated protein kinase-dependent manner

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5-amino-4-imidazolecarboxamide riboside confers strong tolerance to glucose starvation in a 5'-AMP-activated protein kinase-dependent fashion

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An inducible pathway for degradation of FLIP protein sensitizes tumor cells to TRAIL-induced apoptosis

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Mechanisms of UV-induced signal transduction

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Involvement of the DNA mismatch repair system in antineoplastic drug resistance

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The role of c-FLIP in modulation of CD95-induced apoptosis

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Apoptosis of adherent cells by recruitment of caspase-8 to unligated integrins

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Identification of a novel protein kinase mediating Akt survival signaling to the ATM protein

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Involvement of MACH, a novel MORT1/FADD-interacting protease, in Fas/APO-1- and TNF receptor-induced cell death

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PKB binding proteins. Getting in on the Akt.

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Regulation of cell death protease caspase-9 by phosphorylation

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FADD, a novel death domain-containing protein, interacts with the death domain of Fas and initiates apoptosis

1 reference

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based on heuristic

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Cellular survival: a play in three Akts

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Akt phosphorylation of BAD couples survival signals to the cell-intrinsic death machinery

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Tumor angiogenesis

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Can mosaic tumor vessels facilitate molecular diagnosis of cancer?

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Mitochondria and apoptosis

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based on heuristic

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Molecular steps of tumor necrosis factor receptor-mediated apoptosis

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Akt mediates Rac/Cdc42-regulated cell motility in growth factor-stimulated cells and in invasive PTEN knockout cells

1 reference

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Inhibition of death receptor signals by cellular FLIP

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Critical roles of AMP-activated protein kinase in constitutive tolerance of cancer cells to nutrient deprivation and tumor formation

1 reference

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Cell death induced by topoisomerase-targeted drugs: more questions than answers

1 reference

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The PI 3-kinase/Akt signaling pathway delivers an anti-apoptotic signal.

1 reference

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Identification and characterization of a novel sucrose-non-fermenting protein kinase/AMP-activated protein kinase-related protein kinase, SNARK

1 reference

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7 January 2021

based on heuristic

inferred from DOI database lookup

Cleavage of BID by caspase 8 mediates the mitochondrial damage in the Fas pathway of apoptosis

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Bid, a Bcl2 interacting protein, mediates cytochrome c release from mitochondria in response to activation of cell surface death receptors

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1206899

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FLICE, a novel FADD-homologous ICE/CED-3-like protease, is recruited to the CD95 (Fas/APO-1) death--inducing signaling complex

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1206899

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based on heuristic

inferred from DOI database lookup

Apoptosis by death factor

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7 January 2021

based on heuristic

inferred from DOI database lookup

Low glucose-enhanced TRAIL cytotoxicity is mediated through the ceramide-Akt-FLIP pathway

1 reference

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7 January 2021

based on heuristic

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An antagonist decoy receptor and a death domain-containing receptor for TRAIL

1 reference

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Control of TRAIL-induced apoptosis by a family of signaling and decoy receptors

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1206899

7 January 2021

based on heuristic

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Mammalian AMP-activated protein kinase subfamily

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based on heuristic

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Viral FLICE-inhibitory proteins (FLIPs) prevent apoptosis induced by death receptors

1 reference

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based on heuristic

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Cloning of the chromosome breakpoint of neoplastic B cells with the t(14;18) chromosome translocation

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Bcl-2 gene promotes haemopoietic cell survival and cooperates with c-myc to immortalize pre-B cells

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BID: a novel BH3 domain-only death agonist

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Chemotherapeutic agents augment TRAIL-induced apoptosis in human hepatocellular carcinoma cell lines

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Identifiers

10.1038/SJ.ONC.1206899

1 reference

Europe PubMed Central

31 July 2017

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1 reference

Europe PubMed Central

31 July 2017

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