(Q34979751)

5 August 2017

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title

Amyloid-beta and tau serve antioxidant functions in the aging and Alzheimer brain (English)

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5 August 2017

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4

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5 August 2017

1

Mark A Smith

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5 August 2017

2

Gemma Casadesus

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5 August 2017

James A Joseph

3

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5 August 2017

publication date

1 November 2002

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Free Radical Biology and Medicine

5 August 2017

published in

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5 August 2017

volume

33

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5 August 2017

issue

9

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5 August 2017

page(s)

1194-1199

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Diagnosis of Alzheimer's disease

5 August 2017

cites work

1 reference

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7 January 2021

The Consortium to Establish a Registry for Alzheimer's Disease (CERAD). Part II. Standardization of the neuropathologic assessment of Alzheimer's disease

1 reference

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7 January 2021

Alzheimer's disease results from the cerebral accumulation and cytotoxicity of amyloid beta-protein

1 reference

https://api.crossref.org/works/10.1016%2FS0891-5849%2802%2901021-3

7 January 2021

Abeta associated neuropil changes: correlation with neuronal loss and dementia

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7 January 2021

Alzheimer's disease: genotypes, phenotypes, and treatments

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7 January 2021

Arson. Tracking the culprit in Alzheimer's disease

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7 January 2021

Neuronal oxidative stress precedes amyloid-beta deposition in Down syndrome

1 reference

https://api.crossref.org/works/10.1016%2FS0891-5849%2802%2901021-3

7 January 2021

Oxidative damage is the earliest event in Alzheimer disease

1 reference

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7 January 2021

Non-enzymatically glycated tau in Alzheimer's disease induces neuronal oxidant stress resulting in cytokine gene expression and release of amyloid beta-peptide.

1 reference

https://api.crossref.org/works/10.1016%2FS0891-5849%2802%2901021-3

7 January 2021

Evidence that the beta-amyloid plaques of Alzheimer's disease represent the redox-silencing and entombment of abeta by zinc

1 reference

https://api.crossref.org/works/10.1016%2FS0891-5849%2802%2901021-3

7 January 2021

Alzheimer neuropathologic alterations in aged cognitively normal subjects

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7 January 2021

1 reference

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7 January 2021

A4 amyloid protein deposition and the diagnosis of Alzheimer's disease: prevalence in aged brains determined by immunocytochemistry compared with conventional neuropathologic techniques.

1 reference

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7 January 2021

Soluble pool of Abeta amyloid as a determinant of severity of neurodegeneration in Alzheimer's disease

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7 January 2021

Enhanced production and oligomerization of the 42-residue amyloid beta-protein by Chinese hamster ovary cells stably expressing mutant presenilins

1 reference

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7 January 2021

Naturally secreted oligomers of amyloid beta protein potently inhibit hippocampal long-term potentiation in vivo

1 reference

https://api.crossref.org/works/10.1016%2FS0891-5849%2802%2901021-3

7 January 2021

Long-term dietary strawberry, spinach, or vitamin E supplementation retards the onset of age-related neuronal signal-transduction and cognitive behavioral deficits.

1 reference

https://api.crossref.org/works/10.1016%2FS0891-5849%2802%2901021-3

7 January 2021

Reversals of age-related declines in neuronal signal transduction, cognitive, and motor behavioral deficits with blueberry, spinach, or strawberry dietary supplementation.

1 reference

https://api.crossref.org/works/10.1016%2FS0891-5849%2802%2901021-3

7 January 2021

Antioxidant-rich diets improve cerebellar physiology and motor learning in aged rats.

1 reference

https://api.crossref.org/works/10.1016%2FS0891-5849%2802%2901021-3

7 January 2021

Overexpression of heme oxygenase in neuronal cells, the possible interaction with Tau

1 reference

https://api.crossref.org/works/10.1016%2FS0891-5849%2802%2901021-3

7 January 2021

What counts in brain aging? Design-based stereological analysis of cell number

1 reference

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7 January 2021

Correlative Memory Deficits, Abeta Elevation, and Amyloid Plaques in Transgenic Mice

1 reference

https://api.crossref.org/works/10.1016%2FS0891-5849%2802%2901021-3

7 January 2021

Amyloid-β Deposition in Alzheimer Transgenic Mice Is Associated with Oxidative Stress

1 reference

https://api.crossref.org/works/10.1016%2FS0891-5849%2802%2901021-3

7 January 2021

Increased lipid peroxidation precedes amyloid plaque formation in an animal model of Alzheimer amyloidosis.

1 reference

https://api.crossref.org/works/10.1016%2FS0891-5849%2802%2901021-3

7 January 2021

Ubiquitin is a component of paired helical filaments in Alzheimer's disease.

1 reference

https://api.crossref.org/works/10.1016%2FS0891-5849%2802%2901021-3

7 January 2021

Ubiquitin is detected in neurofibrillary tangles and senile plaque neurites of Alzheimer disease brains

1 reference

https://api.crossref.org/works/10.1016%2FS0891-5849%2802%2901021-3

7 January 2021

Redox-active iron mediates amyloid-beta toxicity

1 reference

https://api.crossref.org/works/10.1016%2FS0891-5849%2802%2901021-3

7 January 2021

beta-Amyloid of Alzheimer's disease induces reactive gliosis that inhibits axonal outgrowth.

1 reference

https://api.crossref.org/works/10.1016%2FS0891-5849%2802%2901021-3

7 January 2021

Astrocytes regulate microglial phagocytosis of senile plaque cores of Alzheimer's disease

1 reference

https://api.crossref.org/works/10.1016%2FS0891-5849%2802%2901021-3

7 January 2021

In vitro and in vivo protein oxidation induced by Alzheimer's disease amyloid beta-peptide (1-42).

1 reference

https://api.crossref.org/works/10.1016%2FS0891-5849%2802%2901021-3

7 January 2021

Amyloid beta-peptide-associated free radical oxidative stress, neurotoxicity, and Alzheimer's disease

1 reference

https://api.crossref.org/works/10.1016%2FS0891-5849%2802%2901021-3

7 January 2021

Alzheimer's disease amyloid beta peptide 25-35 inhibits lipid peroxidation as a result of its membrane interactions.

1 reference

https://api.crossref.org/works/10.1016%2FS0891-5849%2802%2901021-3

7 January 2021

Neuron loss in APP transgenic mice

1 reference

https://api.crossref.org/works/10.1016%2FS0891-5849%2802%2901021-3

7 January 2021

Correlation between cognitive deficits and Abeta deposits in transgenic APP+PS1 mice

1 reference

https://api.crossref.org/works/10.1016%2FS0891-5849%2802%2901021-3

7 January 2021

Neuronal loss correlates with but exceeds neurofibrillary tangles in Alzheimer's disease.

1 reference

https://api.crossref.org/works/10.1016%2FS0891-5849%2802%2901021-3

7 January 2021

Neuron loss from the hippocampus of Alzheimer's disease exceeds extracellular neurofibrillary tangle formation

1 reference

https://api.crossref.org/works/10.1016%2FS0891-5849%2802%2901021-3

7 January 2021

RNA oxidation is a prominent feature of vulnerable neurons in Alzheimer's disease.

1 reference

https://api.crossref.org/works/10.1016%2FS0891-5849%2802%2901021-3

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0891-5849%2802%2901021-3

7 January 2021

In Alzheimer's disease, heme oxygenase is coincident with Alz50, an epitope of tau induced by 4-hydroxy-2-nonenal modification.

1 reference

https://api.crossref.org/works/10.1016%2FS0891-5849%2802%2901021-3

7 January 2021

Protein-bound acrolein: a novel marker of oxidative stress in Alzheimer's disease.

1 reference

https://api.crossref.org/works/10.1016%2FS0891-5849%2802%2901021-3

7 January 2021

Neurons may live for decades with neurofibrillary tangles

1 reference

https://api.crossref.org/works/10.1016%2FS0891-5849%2802%2901021-3

7 January 2021

High molecular weight neurofilament proteins are physiological substrates of adduction by the lipid peroxidation product hydroxynonenal

1 reference

https://api.crossref.org/works/10.1016%2FS0891-5849%2802%2901021-3

7 January 2021

Activation of p38 kinase links tau phosphorylation, oxidative stress, and cell cycle-related events in Alzheimer disease

1 reference

https://api.crossref.org/works/10.1016%2FS0891-5849%2802%2901021-3

7 January 2021

Differential activation of neuronal ERK, JNK/SAPK and p38 in Alzheimer disease: the 'two hit' hypothesis

1 reference

https://api.crossref.org/works/10.1016%2FS0891-5849%2802%2901021-3

7 January 2021

Activation and redistribution of c-jun N-terminal kinase/stress activated protein kinase in degenerating neurons in Alzheimer's disease.

1 reference

https://api.crossref.org/works/10.1016%2FS0891-5849%2802%2901021-3

7 January 2021

Activation of MKK6, an upstream activator of p38, in Alzheimer's disease

1 reference

https://api.crossref.org/works/10.1016%2FS0891-5849%2802%2901021-3

7 January 2021

Lipoperoxidation is selectively involved in progressive supranuclear palsy

1 reference

https://api.crossref.org/works/10.1016%2FS0891-5849%2802%2901021-3

7 January 2021

Role of oxidative stress in frontotemporal dementia

1 reference

https://api.crossref.org/works/10.1016%2FS0891-5849%2802%2901021-3

7 January 2021

Phosphorylation of tau alters its association with the plasma membrane

1 reference

https://api.crossref.org/works/10.1016%2FS0891-5849%2802%2901021-3

7 January 2021

https://api.crossref.org/works/10.1016%2FS0891-5849%2802%2901021-3

1 reference

7 January 2021

Predicting the failure of amyloid-beta vaccine.

1 reference

https://api.crossref.org/works/10.1016%2FS0891-5849%2802%2901021-3

7 January 2021

The state versus amyloid-beta: the trial of the most wanted criminal in Alzheimer disease

1 reference

https://api.crossref.org/works/10.1016%2FS0891-5849%2802%2901021-3

7 January 2021

10.1016/S0891-5849(02)01021-3

Identifiers

DOI

1 reference

5 August 2017

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