(Q36795890)

English

Selective inhibition of alpha/beta-hydrolase domain 6 attenuates neurodegeneration, alleviates blood brain barrier breakdown, and improves functional recovery in a mouse model of traumatic brain injury

scientific article published on 5 April 2013

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scholarly article

1 reference

Europe PubMed Central

16 August 2017

Selective inhibition of alpha/beta-hydrolase domain 6 attenuates neurodegeneration, alleviates blood brain barrier breakdown, and improves functional recovery in a mouse model of traumatic brain injury (English)

1 reference

Europe PubMed Central

16 August 2017

traumatic brain injury

0 references

blood–brain barrier

1 reference

based on heuristic

inferred from title

neurodegeneration

1 reference

based on heuristic

inferred from title

author name string

Flaubert Tchantchou

1

1 reference

Europe PubMed Central

16 August 2017

Yumin Zhang

2

1 reference

Europe PubMed Central

16 August 2017

publication date

5 April 2013

1 reference

Europe PubMed Central

16 August 2017

Journal of Neurotrauma

1 reference

Europe PubMed Central

16 August 2017

30

1 reference

Europe PubMed Central

16 August 2017

7

1 reference

Europe PubMed Central

16 August 2017

565-579

1 reference

Europe PubMed Central

16 August 2017

describes a project that uses

1 reference

Europe PubMed Central

11 June 2022

https://www.ebi.ac.uk/europepmc/webservices/rest/PMC3636589/fullTextXML

based on heuristic

inferred from PubMed Central ID database lookup

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1 reference

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Lithium ameliorates neurodegeneration, suppresses neuroinflammation, and improves behavioral performance in a mouse model of traumatic brain injury

1 reference

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Dual inhibition of alpha/beta-hydrolase domain 6 and fatty acid amide hydrolase increases endocannabinoid levels in neurons.

1 reference

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1 reference

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The serine hydrolase ABHD6 controls the accumulation and efficacy of 2-AG at cannabinoid receptors

1 reference

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Valproate administered after traumatic brain injury provides neuroprotection and improves cognitive function in rats

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Administration of S-nitrosoglutathione after traumatic brain injury protects the neurovascular unit and reduces secondary injury in a rat model of controlled cortical impact

1 reference

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1 reference

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Enhancement of endocannabinoid signaling by fatty acid amide hydrolase inhibition: a neuroprotective therapeutic modality

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Genetic disruption of cyclooxygenase-2 does not improve histological or behavioral outcome after traumatic brain injury in mice

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based on heuristic

inferred from PubMed ID database lookup

Identifiers

10.1089/NEU.2012.2647

1 reference

Europe PubMed Central

16 August 2017

1 reference

Europe PubMed Central

16 August 2017

1 reference

Europe PubMed Central

16 August 2017

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