(Q45292219)

11 December 2017

title

Specific caspase interactions and amplification are involved in selective neuronal vulnerability in Huntington's disease (English)

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11 December 2017

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16

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11 December 2017

18

L M Ellerby

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11 December 2017

E Hermel

1

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11 December 2017

J Gafni

2

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11 December 2017

S S Propp

3

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11 December 2017

B R Leavitt

4

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11 December 2017

C L Wellington

5

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11 December 2017

J E Young

6

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11 December 2017

A S Hackam

7

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11 December 2017

A V Logvinova

8

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11 December 2017

A L Peel

9

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11 December 2017

S F Chen

10

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11 December 2017

V Hook

11

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11 December 2017

R Singaraja

12

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11 December 2017

S Krajewski

13

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11 December 2017

P C Goldsmith

14

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11 December 2017

H M Ellerby

15

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11 December 2017

D E Bredesen

17

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11 December 2017

publication date

1 April 2004

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Cell Death & Differentiation

11 December 2017

published in

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11 December 2017

volume

11

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11 December 2017

issue

4

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11 December 2017

page(s)

424-438

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https://scigraph.springernature.com/pub.10.1038/sj.cdd.4401358

11 December 2017

exact match

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cites work

A novel gene containing a trinucleotide repeat that is expanded and unstable on Huntington's disease chromosomes.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401358

7 January 2021

Cloning of the gene for spinocerebellar ataxia 2 reveals a locus with high sensitivity to expanded CAG/glutamine repeats

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https://api.crossref.org/works/10.1038%2FSJ.CDD.4401358

7 January 2021

CAG expansions in a novel gene for Machado-Joseph disease at chromosome 14q32.1

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401358

7 January 2021

Unstable expansion of CAG repeat in hereditary dentatorubral-pallidoluysian atrophy (DRPLA)

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https://api.crossref.org/works/10.1038%2FSJ.CDD.4401358

7 January 2021

Androgen receptor gene mutations in X-linked spinal and bulbar muscular atrophy

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401358

7 January 2021

An expanded CAG repeat sequence in spinocerebellar ataxia type 7

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401358

7 January 2021

Dentatorubral and pallidoluysian atrophy expansion of an unstable CAG trinucleotide on chromosome 12p

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401358

7 January 2021

Expansion of an unstable trinucleotide CAG repeat in spinocerebellar ataxia type 1.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401358

7 January 2021

Moderate expansion of a normally biallelic trinucleotide repeat in spinocerebellar ataxia type 2.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401358

7 January 2021

Identification of the spinocerebellar ataxia type 2 gene using a direct identification of repeat expansion and cloning technique, DIRECT.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401358

7 January 2021

Autosomal dominant cerebellar ataxia (SCA6) associated with small polyglutamine expansions in the alpha 1A-voltage-dependent calcium channel

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https://api.crossref.org/works/10.1038%2FSJ.CDD.4401358

7 January 2021

When more is less: pathogenesis of glutamine repeat neurodegenerative diseases

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https://api.crossref.org/works/10.1038%2FSJ.CDD.4401358

7 January 2021

Huntington's disease. Pathogenesis and management

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401358

7 January 2021

Neuropathological classification of Huntington's disease

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401358

7 January 2021

Identification and localization of huntingtin in brain and human lymphoblastoid cell lines with anti-fusion protein antibodies

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401358

7 January 2021

Widespread expression of Huntington's disease gene (IT15) protein product

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401358

7 January 2021

Polyglutamine expansion as a pathological epitope in Huntington's disease and four dominant cerebellar ataxias

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401358

7 January 2021

Subcellular localization of the Huntington's disease gene product in cell lines by immunofluorescence and biochemical subcellular fractionation

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401358

7 January 2021

Huntingtin is a cytoplasmic protein associated with vesicles in human and rat brain neurons

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401358

7 January 2021

Evidence for degenerative and regenerative changes in neostriatal spiny neurons in Huntington's disease

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401358

7 January 2021

Differential loss of striatal projection neurons in Huntington disease

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401358

7 January 2021

Evidence for a preferential loss of enkephalin immunoreactivity in the external globus pallidus in low grade Huntington's disease using high resolution image analysis

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401358

7 January 2021

Evidence for apoptotic cell death in Huntington disease and excitotoxic animal models.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401358

7 January 2021

Cleavage of atrophin-1 at caspase site aspartic acid 109 modulates cytotoxicity

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401358

7 January 2021

Kennedy's disease: caspase cleavage of the androgen receptor is a crucial event in cytotoxicity

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401358

7 January 2021

Cleavage of huntingtin by apopain, a proapoptotic cysteine protease, is modulated by the polyglutamine tract

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401358

7 January 2021

Caspase cleavage of gene products associated with triplet expansion disorders generates truncated fragments containing the polyglutamine tract

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401358

7 January 2021

A YAC mouse model for Huntington's disease with full-length mutant huntingtin, cytoplasmic toxicity, and selective striatal neurodegeneration.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401358

7 January 2021

Caspase cleavage of mutant huntingtin precedes neurodegeneration in Huntington's disease.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401358

7 January 2021

Calpain activation in Huntington's disease.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401358

7 January 2021

Loss of huntingtin-mediated BDNF gene transcription in Huntington's disease

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401358

7 January 2021

The influence of huntingtin protein size on nuclear localization and cellular toxicity.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401358

7 January 2021

Length of huntingtin and its polyglutamine tract influences localization and frequency of intracellular aggregates

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401358

7 January 2021

Inhibiting caspase cleavage of huntingtin reduces toxicity and aggregate formation in neuronal and nonneuronal cells

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401358

7 January 2021

Death in the balance: alternative participation of the caspase-2 and -9 pathways in neuronal death induced by nerve growth factor deprivation.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401358

7 January 2021

Caspase-8 is required for cell death induced by expanded polyglutamine repeats.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401358

7 January 2021

Release of caspase-9 from mitochondria during neuronal apoptosis and cerebral ischemia

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https://api.crossref.org/works/10.1038%2FSJ.CDD.4401358

7 January 2021

Caspase-2 is localized at the Golgi complex and cleaves golgin-160 during apoptosis

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401358

7 January 2021

Coupling endoplasmic reticulum stress to the cell death program: role of the ER chaperone GRP78

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401358

7 January 2021

Inhibition of nuclear transport of caspase-7 by its prodomain

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401358

7 January 2021

Expression of extended polyglutamine sequentially activates initiator and effector caspases

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401358

7 January 2021

Amino-terminal fragments of mutant huntingtin show selective accumulation in striatal neurons and synaptic toxicity

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401358

7 January 2021

Selective striatal neuronal loss in a YAC128 mouse model of Huntington disease

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401358

7 January 2021

Tissue-specific proteolysis of Huntingtin (htt) in human brain: evidence of enhanced levels of N- and C-terminal htt fragments in Huntington's disease striatum.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401358

7 January 2021

Huntingtin acts in the nucleus to induce apoptosis but death does not correlate with the formation of intranuclear inclusions.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401358

7 January 2021

Identifiers

DOI

10.1038/SJ.CDD.4401358

1 reference

Dimensions Publication ID

11 December 2017

1049670044

0 references

1 reference