(Q45423209)

14 December 2017

title

GSK-3α/β kinases and amyloid production in vivo (English)

1 reference

14 December 2017

11

1 reference

14 December 2017

Tomasz Jaworski

1

0 references

author name string

Ilse Dewachter

2

1 reference

14 December 2017

Benoit Lechat

3

1 reference

14 December 2017

Maarten Gees

4

1 reference

14 December 2017

Anna Kremer

5

1 reference

14 December 2017

David Demedts

6

1 reference

14 December 2017

Peter Borghgraef

7

1 reference

14 December 2017

Herman Devijver

8

1 reference

14 December 2017

Seb Kügler

9

1 reference

14 December 2017

Satish Patel

10

1 reference

14 December 2017

Fred Van Leuven

12

1 reference

14 December 2017

language of work or name

English

0 references

publication date

7 December 2011

1 reference

14 December 2017

1 reference

14 December 2017

volume

480

1 reference

14 December 2017

issue

7376

1 reference

14 December 2017

page(s)

E4-5; discussion E6

1 reference

https://scigraph.springernature.com/pub.10.1038/nature10615

14 December 2017

exact match

0 references

cites work

GSK-3alpha regulates production of Alzheimer's disease amyloid-beta peptides

1 reference

https://api.crossref.org/works/10.1038%2FNATURE10615

Glycogen synthase kinase-3beta phosphorylates protein tau and rescues the axonopathy in the central nervous system of human four-repeat tau transgenic mice.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FNATURE10615

Amyloid activates GSK-3beta to aggravate neuronal tauopathy in bigenic mice

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FNATURE10615

Abnormalities in brain structure and behavior in GSK-3alpha mutant mice

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FNATURE10615

Light and electron microscopy study of glycogen synthase kinase-3beta in the mouse brain

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FNATURE10615

AAV-tau mediates pyramidal neurodegeneration by cell-cycle re-entry without neurofibrillary tangle formation in wild-type mice

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FNATURE10615

Deficiency of presenilin-1 inhibits the normal cleavage of amyloid precursor protein

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FNATURE10615

Early phenotypic changes in transgenic mice that overexpress different mutants of amyloid precursor protein in brain

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FNATURE10615

Alzheimer's disease: old problem, new views from transgenic and viral models

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FNATURE10615

Neuronal deficiency of presenilin 1 inhibits amyloid plaque formation and corrects hippocampal long-term potentiation but not a cognitive defect of amyloid precursor protein [V717I] transgenic mice.

21 January 2018

1 reference

12 December 2020

Pathological Hallmarks, Clinical Parallels, and Value for Drug Testing in Alzheimer's Disease of the APP[V717I] London Transgenic Mouse Model

1 reference

12 December 2020

GSK-3 is essential in the pathogenesis of Alzheimer's disease.

1 reference

12 December 2020

Glycogen synthase kinase 3β is identical to tau protein kinase I generating several epitopes of paired helical filaments

1 reference

12 December 2020

Identifiers

DOI

10.1038/NATURE10615

1 reference

Dimensions Publication ID

14 December 2017

1027089927

0 references

1 reference