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Ripped to death
scientific article
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scholarly article
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review article
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stated in
Europe PubMed Central
title
Ripped to death
(English)
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main subject
cell biology
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author
Ricardo Weinlich
object named as
Ricardo Weinlich
series ordinal
1
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Douglas R. Green
object named as
Douglas R Green
series ordinal
3
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author name string
Christopher P Dillon
series ordinal
2
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language of work or name
English
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publication date
November 2011
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published in
Trends in Cell Biology
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volume
21
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issue
11
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page(s)
630-7
0 references
cites work
cIAPs block Ripoptosome formation, a RIP1/caspase-8 containing intracellular cell death complex differentially regulated by cFLIP isoforms
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NEMO and RIP1 control cell fate in response to extensive DNA damage via TNF-α feedforward signaling
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SHARPIN forms a linear ubiquitin ligase complex regulating NF-κB activity and apoptosis
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Recruitment of the linear ubiquitin chain assembly complex stabilizes the TNF-R1 signaling complex and is required for TNF-mediated gene induction
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Phosphorylation-driven assembly of the RIP1-RIP3 complex regulates programmed necrosis and virus-induced inflammation
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Receptor interacting protein kinase-3 determines cellular necrotic response to TNF-alpha
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TNF-alpha induces two distinct caspase-8 activation pathways
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The Fas-associated death domain protein is required in apoptosis and TLR-induced proliferative responses in B cells
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Fas triggers an alternative, caspase-8-independent cell death pathway using the kinase RIP as effector molecule
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Cleavage of the death domain kinase RIP by caspase-8 prompts TNF-induced apoptosis
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Apaf1 (CED-4 homolog) regulates programmed cell death in mammalian development
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Cleavage of BID by caspase 8 mediates the mitochondrial damage in the Fas pathway of apoptosis
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Inhibition of death receptor signals by cellular FLIP
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The TNF receptor 1-associated protein TRADD signals cell death and NF-kappa B activation
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7 April 2017
Phosphorylation of FADD at serine 194 by CKIalpha regulates its nonapoptotic activities
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Essential roles of Atg5 and FADD in autophagic cell death: dissection of autophagic cell death into vacuole formation and cell death
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Caspase-8 serves both apoptotic and nonapoptotic roles
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Ex vivo whole-embryo culture of caspase-8-deficient embryos normalize their aberrant phenotypes in the developing neural tube and heart
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The long form of FLIP is an activator of caspase-8 at the Fas death-inducing signaling complex
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7 April 2017
Cellular FLICE-inhibitory protein splice variants inhibit different steps of caspase-8 activation at the CD95 death-inducing signaling complex
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7 April 2017
Apaf1 is required for mitochondrial pathways of apoptosis and brain development
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7 April 2017
Fas-mediated apoptosis and activation-induced T-cell proliferation are defective in mice lacking FADD/Mort1
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FADD: essential for embryo development and signaling from some, but not all, inducers of apoptosis
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FLICE is activated by association with the CD95 death-inducing signaling complex (DISC).
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7 April 2017
Mice deficient for the 55 kd tumor necrosis factor receptor are resistant to endotoxic shock, yet succumb to L. monocytogenes infection
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7 April 2017
Embryonic lethality and liver degeneration in mice lacking the RelA component of NF-kappa B
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7 April 2017
Caspase 8 inhibits programmed necrosis by processing CYLD
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7 April 2017
Mechanisms of necroptosis in T cells
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7 April 2017
Catalytic activity of the caspase-8-FLIP(L) complex inhibits RIPK3-dependent necrosis
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7 April 2017
RIP3, an energy metabolism regulator that switches TNF-induced cell death from apoptosis to necrosis
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7 April 2017
Insights into the regulatory mechanism for caspase-8 activation
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7 April 2017
RIP3 mediates the embryonic lethality of caspase-8-deficient mice
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7 April 2017
Identification of RIP1 kinase as a specific cellular target of necrostatins
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7 April 2017
Chemical inhibitor of nonapoptotic cell death with therapeutic potential for ischemic brain injury
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7 April 2017
Molecular mechanisms of caspase regulation during apoptosis
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7 April 2017
Reduced apoptosis and cytochrome c-mediated caspase activation in mice lacking caspase 9
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7 April 2017
The death domain kinase RIP mediates the TNF-induced NF-kappaB signal
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7 April 2017
Suppression of TNF-alpha-induced apoptosis by NF-kappaB
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7 April 2017
TNF- and cancer therapy-induced apoptosis: potentiation by inhibition of NF-kappaB
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7 April 2017
The Ripoptosome, a signaling platform that assembles in response to genotoxic stress and loss of IAPs
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27 September 2017
Functional complementation between FADD and RIP1 in embryos and lymphocytes
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27 September 2017
FLIP(L) induces caspase 8 activity in the absence of interdomain caspase 8 cleavage and alters substrate specificity
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27 September 2017
Fas-associated death domain (FADD) is a negative regulator of T-cell receptor-mediated necroptosis
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27 September 2017
Virus inhibition of RIP3-dependent necrosis
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27 September 2017
Inducible dimerization and inducible cleavage reveal a requirement for both processes in caspase-8 activation
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27 September 2017
Tumor necrosis factor-mediated cell death: to break or to burst, that's the question
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27 September 2017
Cellular IAPs inhibit a cryptic CD95-induced cell death by limiting RIP1 kinase recruitment
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27 September 2017
Vaccinia virus protein F1L is a caspase-9 inhibitor
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27 September 2017
Necroptosis, necrosis and secondary necrosis converge on similar cellular disintegration features.
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27 September 2017
Antigen-mediated T cell expansion regulated by parallel pathways of death
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27 September 2017
Beyond tumor necrosis factor receptor: TRADD signaling in toll-like receptors.
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27 September 2017
Mutation of a self-processing site in caspase-8 compromises its apoptotic but not its nonapoptotic functions in bacterial artificial chromosome-transgenic mice
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27 September 2017
The function of TRADD in signaling through tumor necrosis factor receptor 1 and TRIF-dependent Toll-like receptors
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27 September 2017
Control of apoptosis by human cytomegalovirus.
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27 September 2017
Cleavage of RIP3 inactivates its caspase-independent apoptosis pathway by removal of kinase domain.
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PubMed Central
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27 September 2017
TNF-induced activation of the Nox1 NADPH oxidase and its role in the induction of necrotic cell death
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27 September 2017
Activation of caspases-8 and -10 by FLIP(L).
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27 September 2017
Viral FLIP impairs survival of activated T cells and generation of CD8+ T cell memory.
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27 September 2017
Kinase RIP3 is dispensable for normal NF-kappa Bs, signaling by the B-cell and T-cell receptors, tumor necrosis factor receptor 1, and Toll-like receptors 2 and 4.
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27 September 2017
Essential role for caspase 8 in T-cell homeostasis and T-cell-mediated immunity
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27 September 2017
NF-kappaB signals induce the expression of c-FLIP.
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27 September 2017
Human herpesvirus 8 viral FLICE-inhibitory protein inhibits Fas-mediated apoptosis through binding and prevention of procaspase-8 maturation.
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PubMed Central
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27 September 2017
T cell-specific FADD-deficient mice: FADD is required for early T cell development
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27 September 2017
Requirement for Casper (c-FLIP) in regulation of death receptor-induced apoptosis and embryonic development
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27 September 2017
Caspase activation is required for T cell proliferation
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27 September 2017
Early activation of caspases during T lymphocyte stimulation results in selective substrate cleavage in nonapoptotic cells
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PubMed Central
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27 September 2017
Caspase-8 is required for cell death induced by expanded polyglutamine repeats.
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PubMed Central
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27 September 2017
Severe liver degeneration in mice lacking the IkappaB kinase 2 gene
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27 September 2017
Differential requirement for caspase 9 in apoptotic pathways in vivo.
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27 September 2017
A dominant interfering mutant of FADD/MORT1 enhances deletion of autoreactive thymocytes and inhibits proliferation of mature T lymphocytes
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PubMed Central
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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3205316
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27 September 2017
Viral FLICE-inhibitory proteins (FLIPs) prevent apoptosis induced by death receptors
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PubMed Central
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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3205316
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27 September 2017
Decreased apoptosis in the brain and premature lethality in CPP32-deficient mice
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27 September 2017
CrmA expression in T lymphocytes of transgenic mice inhibits CD95 (Fas/APO-1)-transduced apoptosis, but does not cause lymphadenopathy or autoimmune disease.
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27 September 2017
Fas and Fas ligand: lpr and gld mutations
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27 September 2017
A new mutation, gld, that produces lymphoproliferation and autoimmunity in C3H/HeJ mice
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27 September 2017
Spontaneous murine lupus-like syndromes. Clinical and immunopathological manifestations in several strains
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27 September 2017
A Function of Fas-Associated Death Domain Protein in Cell Cycle Progression Localized to a Single Amino Acid at Its C-Terminal Region
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PubMed Central
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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3205316
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30 May 2018
Anti-apoptotic oncogenes prevent caspase-dependent and independent commitment for cell death
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30 May 2018
Targeted disruption of the mouse Caspase 8 gene ablates cell death induction by the TNF receptors, Fas/Apo1, and DR3 and is lethal prenatally.
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30 May 2018
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30 May 2018
Inhibition of caspases increases the sensitivity of L929 cells to necrosis mediated by tumor necrosis factor.
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30 May 2018
Arachidonic acid-selective cytosolic phospholipase A2 is crucial in the cytotoxic action of tumor necrosis factor.
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30 May 2018
NF-kappaB suppression by the deubiquitinating enzyme Cezanne: a novel negative feedback loop in pro-inflammatory signaling
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28 November 2018
Role of proteolysis in caspase-8 activation and stabilization
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PubMed Central
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28 November 2018
Caspase inhibition blocks human T cell proliferation by suppressing appropriate regulation of IL-2, CD25, and cell cycle-associated proteins
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28 November 2018
The kinase activity of Rip1 is not required for tumor necrosis factor-alpha-induced IkappaB kinase or p38 MAP kinase activation or for the ubiquitination of Rip1 by Traf2.
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PubMed Central
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28 November 2018
Tumor necrosis factor-induced nonapoptotic cell death requires receptor-interacting protein-mediated cellular reactive oxygen species accumulation
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28 November 2018
The requirements for Fas-associated death domain signaling in mature T cell activation and survival
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28 November 2018
Fas-associated Death Domain Protein and Caspase-8 Are Not Recruited to the Tumor Necrosis Factor Receptor 1 Signaling Complex during Tumor Necrosis Factor-induced Apoptosis
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28 November 2018
Accelerated degradation of cellular FLIP protein through the ubiquitin-proteasome pathway in p53-mediated apoptosis of human cancer cells
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28 November 2018
Differential activation of phospholipases during necrosis or apoptosis: a comparative study using tumor necrosis factor and anti-Fas antibodies.
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28 November 2018
Targeted mutation in the Fas gene causes hyperplasia in peripheral lymphoid organs and liver
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12 December 2020
based on heuristic
inferred from PubMed ID database lookup
Inhibition of caspase activity induces a switch from apoptosis to necrosis
1 reference
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PubMed
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https://pubmed.ncbi.nlm.nih.gov/21978761
retrieved
12 December 2020
based on heuristic
inferred from PubMed ID database lookup
Activation of a pro-apoptotic amplification loop through inhibition of NF-kappaB-dependent survival signals by caspase-mediated inactivation of RIP
1 reference
stated in
PubMed
reference URL
https://pubmed.ncbi.nlm.nih.gov/21978761
retrieved
12 December 2020
based on heuristic
inferred from PubMed ID database lookup
Function of TRADD in tumor necrosis factor receptor 1 signaling and in TRIF-dependent inflammatory responses
1 reference
stated in
PubMed
reference URL
https://pubmed.ncbi.nlm.nih.gov/21978761
retrieved
12 December 2020
based on heuristic
inferred from PubMed ID database lookup
Differential activation of phospholipases during necrosis or apoptosis: A comparative study using tumor necrosis factor and anti-Fas antibodies
1 reference
stated in
Crossref
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retrieved
7 January 2021
based on heuristic
inferred from DOI database lookup
Identifiers
DOI
10.1016/J.TCB.2011.09.002
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
2200908
OpenCitations bibliographic resource ID
2200908
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
2200908
PMCID
3205316
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
2200908
PubMed ID
21978761
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
2200908
ResearchGate publication ID
51698865
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