(Q37164157)

English

Caveolin-1 (P132L), a common breast cancer mutation, confers mammary cell invasiveness and defines a novel stem cell/metastasis-associated gene signature

scientific article published on May 2009

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scholarly article

1 reference

Europe PubMed Central

PMC publication ID

18 August 2017

Caveolin-1 (P132L), a common breast cancer mutation, confers mammary cell invasiveness and defines a novel stem cell/metastasis-associated gene signature (English)

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Europe PubMed Central

PMC publication ID

18 August 2017

neoplastic invasiveness

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Federica Sotgia

3

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Federica Sotgia

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Europe PubMed Central

18 August 2017

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Michael P. Lisanti

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Michael P Lisanti

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Richard Pestell

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Richard G Pestell

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18 August 2017

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Chenguang Wang

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18 August 2017

Mathew C Casimiro

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Mathew C Casimiro

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18 August 2017

Philippe G. Frank

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Philippe G Frank

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Neal Flomenberg

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Neal Flomenberg

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Gloria Bonuccelli

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Gloria Bonuccelli

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Kristin M. Daumer

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Kristin M Daumer

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Janet N. Milliman

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Janet N Milliman

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Franco Capozza

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Manran Liu

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Sanjay Katiyar

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Elliott Dew

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Jie Zhou

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Carlo Minetti

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Fabien Alpy

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Marie-Christine Rio

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Catherine Tomasetto

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Isabelle Mercier

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publication date

1 May 2009

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18 August 2017

The American Journal of Pathology

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18 August 2017

174

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18 August 2017

5

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18 August 2017

1650-1662

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18 August 2017

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Europe PubMed Central

22 June 2022

https://europepmc.org/article/PMC/2671254

based on heuristic

inferred from PubMed Central ID database lookup

Musashi1 modulates mammary progenitor cell expansion through proliferin-mediated activation of the Wnt and Notch pathways

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Caveolin-1 mutations in human breast cancer: functional association with estrogen receptor alpha-positive status

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Genes associated with breast cancer metastatic to bone

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Give lipids a START: the StAR-related lipid transfer (START) domain in mammals.

1 reference

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Caveolin-1 in oncogenic transformation, cancer, and metastasis

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Caveolin-1 in breast cancer

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Canonical WNT signaling promotes mammary placode development and is essential for initiation of mammary gland morphogenesis

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Association of the breast cancer protein MLN51 with the exon junction complex via its speckle localizer and RNA binding module

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Caveolin-1 mutations (P132L and null) and the pathogenesis of breast cancer: caveolin-1 (P132L) behaves in a dominant-negative manner and caveolin-1 (-/-) null mice show mammary epithelial cell hyperplasia

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3 September 2018

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1 reference

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A relevant immunomagnetic assay to detect and characterize epithelial cell adhesion molecule-positive cells in bone marrow from patients with breast carcinoma: immunomagnetic purification of micrometastases

1 reference

https://pubmed.ncbi.nlm.nih.gov/19395651

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Expression of tenascin in invasion border of early breast cancer correlates with higher risk of distant metastasis

1 reference

https://pubmed.ncbi.nlm.nih.gov/19395651

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Expression of tenascin-C in intraductal carcinoma of human breast: relationship to invasion

1 reference

https://pubmed.ncbi.nlm.nih.gov/19395651

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Heparan Sulfate-modified CD44 Promotes Hepatocyte Growth Factor/Scatter Factor-induced Signal Transduction through the Receptor Tyrosine Kinase c-Met

1 reference

https://pubmed.ncbi.nlm.nih.gov/19395651

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

High Ep-CAM expression is associated with poor prognosis in node-positive breast cancer

1 reference

https://pubmed.ncbi.nlm.nih.gov/19395651

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Identifiers

10.2353/AJPATH.2009.080648

1 reference

Europe PubMed Central

PMC publication ID

18 August 2017

PMC publication ID

1 reference

Europe PubMed Central

PMC publication ID

18 August 2017

PubMed publication ID

1 reference

Europe PubMed Central

PMC publication ID

18 August 2017

ResearchGate publication ID

0 references

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