(Q38041423)

27 August 2017

title

The selective BH4-domain biology of Bcl-2-family members: IP3Rs and beyond (English)

1 reference

27 August 2017

Giovanni Monaco

1

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27 August 2017

Tim Vervliet

series ordinal

2

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27 August 2017

Haidar Akl

series ordinal

3

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27 August 2017

Geert Bultynck

series ordinal

4

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27 August 2017

publication date

6 September 2012

1 reference

Cellular and Molecular Life Sciences

27 August 2017

published in

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27 August 2017

volume

70

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27 August 2017

issue

7

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27 August 2017

page(s)

1171-1183

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https://scigraph.springernature.com/pub.10.1007/s00018-012-1118-y

27 August 2017

exact match

0 references

cites work

How do BCL-2 proteins induce mitochondrial outer membrane permeabilization?

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Control of mitochondrial apoptosis by the Bcl-2 family

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

21 January 2018

The BCL-2 family reunion

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Bcl-x(L) retrotranslocates Bax from the mitochondria into the cytosol

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Traveling Bax and forth from mitochondria to control apoptosis.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Stepwise activation of BAX and BAK by tBID, BIM, and PUMA initiates mitochondrial apoptosis.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

BAX and BAK caught in the act.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Genetic dissection of the permeability transition pore

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Calcium elevation in mitochondria is the main Ca2+ requirement for mitochondrial permeability transition pore (mPTP) opening.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Bad targets the permeability transition pore independent of Bax or Bak to switch between Ca2+-dependent cell survival and death

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

BH3 profiling identifies three distinct classes of apoptotic blocks to predict response to ABT-737 and conventional chemotherapeutic agents

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Mitochondria primed by death signals determine cellular addiction to antiapoptotic BCL-2 family members

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Differential targeting of prosurvival Bcl-2 proteins by their BH3-only ligands allows complementary apoptotic function

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

BH3 domains of BH3-only proteins differentially regulate Bax-mediated mitochondrial membrane permeabilization both directly and indirectly

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Development and maintenance of B and T lymphocytes requires antiapoptotic MCL-1.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Chronic lymphocytic leukemia requires BCL2 to sequester prodeath BIM, explaining sensitivity to BCL2 antagonist ABT-737.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

BH3 mimetics to improve cancer therapy; mechanisms and examples

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Discovery of an orally bioavailable small molecule inhibitor of prosurvival B-cell lymphoma 2 proteins

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

ABT-263: a potent and orally bioavailable Bcl-2 family inhibitor

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Evidence that BCL-2 represses apoptosis by regulating endoplasmic reticulum-associated Ca2+ fluxes

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Bcl-2 on the endoplasmic reticulum: protecting the mitochondria from a distance

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Bcl-2 protein family members: versatile regulators of calcium signaling in cell survival and apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Bcl-2 and Ca2+ homeostasis in the endoplasmic reticulum

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Calcium and cell death mechanisms: a perspective from the cell death community

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Ca2+ signaling, mitochondria and cell death

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Calcium and apoptosis: ER-mitochondria Ca2+ transfer in the control of apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Structural and functional link between the mitochondrial network and the endoplasmic reticulum

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Ca(2+) transfer from the ER to mitochondria: when, how and why

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

PERK is required at the ER-mitochondrial contact sites to convey apoptosis after ROS-based ER stress

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

The Ca2+ concentration of the endoplasmic reticulum is a key determinant of ceramide-induced apoptosis: significance for the molecular mechanism of Bcl-2 action

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Essential regulation of cell bioenergetics by constitutive InsP3 receptor Ca2+ transfer to mitochondria.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Mechanism of ER stress-induced brain damage by IP(3) receptor

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

The inositol 1,4,5-trisphosphate receptor regulates autophagy through its interaction with Beclin 1.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Ins(1,4,5)P3 receptor-mediated Ca2+ signaling and autophagy induction are interrelated

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

A dual role for Ca(2+) in autophagy regulation

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

VDAC1 selectively transfers apoptotic Ca2+ signals to mitochondria.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Integrative genomics identifies MCU as an essential component of the mitochondrial calcium uniporter

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

A forty-kilodalton protein of the inner membrane is the mitochondrial calcium uniporter

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Studying mitochondrial Ca(2+) uptake - a revisit.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Intracellular Ca2+ storage in health and disease: a dynamic equilibrium.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

The IP(3) receptor-mitochondria connection in apoptosis and autophagy

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

IP(3) Receptors, Mitochondria, and Ca Signaling: Implications for Aging

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Selective modulation of subtype III IP₃R by Akt regulates ER Ca²⁺ release and apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Phosphorylation of inositol 1,4,5-trisphosphate receptors by protein kinase B/Akt inhibits Ca2+ release and apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

The PKB/AKT pathway in cancer

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

PML regulates apoptosis at endoplasmic reticulum by modulating calcium release

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Ca²(+) transfer from the ER to mitochondria: channeling cell death by a tumor suppressor.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

The role of PML in the control of apoptotic cell fate: a new key player at ER-mitochondria sites

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Intramitochondrial calcium regulation by the FHIT gene product sensitizes to apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Voltage-dependent anion channel 1-based peptides interact with Bcl-2 to prevent antiapoptotic activity

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Bcl-2-mediated alterations in endoplasmic reticulum Ca2+ analyzed with an improved genetically encoded fluorescent sensor

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Bax Inhibitor-1 Is a pH-dependent regulator of Ca2+ channel activity in the endoplasmic reticulum

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

BI-1 regulates endoplasmic reticulum Ca2+ homeostasis downstream of Bcl-2 family proteins

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

The C terminus of Bax inhibitor-1 forms a Ca2+-permeable channel pore.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Bax inhibitor-1 is a novel IP₃ receptor-interacting and -sensitizing protein

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Endoplasmic reticulum protein BI-1 regulates Ca²⁺-mediated bioenergetics to promote autophagy

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Reduced loading of intracellular Ca(2+) stores and downregulation of capacitative Ca(2+) influx in Bcl-2-overexpressing cells.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

BAX and BAK regulation of endoplasmic reticulum Ca2+: a control point for apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Proapoptotic BAX and BAK regulate the type 1 inositol trisphosphate receptor and calcium leak from the endoplasmic reticulum

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Maintenance of calcium homeostasis in the endoplasmic reticulum by Bcl-2

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Domoic acid enhances Bcl-2-calcineurin-inositol-1,4,5-trisphosphate receptor interactions and delayed neuronal death in rat brain slices

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Calcium-dependent interaction of calcineurin with Bcl-2 in neuronal tissue

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Bcl-2 functionally interacts with inositol 1,4,5-trisphosphate receptors to regulate calcium release from the ER in response to inositol 1,4,5-trisphosphate.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

The endoplasmic reticulum gateway to apoptosis by Bcl-X(L) modulation of the InsP3R

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Apoptosis protection by Mcl-1 and Bcl-2 modulation of inositol 1,4,5-trisphosphate receptor-dependent Ca2+ signaling

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Bcl-X(L) affects Ca(2+) homeostasis by altering expression of inositol 1,4,5-trisphosphate receptors

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Modulation of endoplasmic reticulum calcium pump by Bcl-2.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Evidence for a role of the sarcoplasmic/endoplasmic reticulum Ca(2+)-ATPase in thapsigargin and Bcl-2 induced changes in Xenopus laevis oocyte maturation

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Praf2 is a novel Bcl-xL/Bcl-2 interacting protein with the ability to modulate survival of cancer cells

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Anti-apoptotic protein Bcl-2 interacts with and destabilizes the sarcoplasmic/endoplasmic reticulum Ca2+-ATPase (SERCA).

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Displacement of SERCA from SR lipid caveolae-related domains by Bcl-2: a possible mechanism for SERCA inactivation

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Heat-shock proteins attenuate SERCA inactivation by the anti-apoptotic protein Bcl-2: possible implications for the ER Ca2+-mediated apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Bcl-2 suppresses sarcoplasmic/endoplasmic reticulum Ca2+-ATPase expression in cystic fibrosis airways: role in oxidant-mediated cell death

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Cell death independent of caspases: a review.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Molecular definitions of cell death subroutines: recommendations of the Nomenclature Committee on Cell Death 2012

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

An alternative, nonapoptotic form of programmed cell death

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Bap31 enhances the endoplasmic reticulum export and quality control of human class I MHC molecules

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

BAP31 interacts with Sec61 translocons and promotes retrotranslocation of CFTRDeltaF508 via the derlin-1 complex

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Uncleaved BAP31 in association with A4 protein at the endoplasmic reticulum is an inhibitor of Fas-initiated release of cytochrome c from mitochondria

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Caspase cleavage product of BAP31 induces mitochondrial fission through endoplasmic reticulum calcium signals, enhancing cytochrome c release to the cytosol

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Bcl2 at the endoplasmic reticulum protects against a Bax/Bak-independent paraptosis-like cell death pathway initiated via p20Bap31.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Ribosome-free terminals of rough ER allow formation of STIM1 puncta and segregation of STIM1 from IP(3) receptors.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

A novel role for Bcl-2 in regulation of cellular calcium extrusion

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Inhibitors of Bcl-2 protein family deplete ER Ca2+ stores in pancreatic acinar cells.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Bcl-2 differentially regulates Ca2+ signals according to the strength of T cell receptor activation

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Bcl-2 suppresses Ca2+ release through inositol 1,4,5-trisphosphate receptors and inhibits Ca2+ uptake by mitochondria without affecting ER calcium store content

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Targeting Bcl-2-IP3 receptor interaction to reverse Bcl-2's inhibition of apoptotic calcium signals

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

The BH4 domain of Bcl-2 inhibits ER calcium release and apoptosis by binding the regulatory and coupling domain of the IP3 receptor

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Bcl-2 interaction with the inositol 1,4,5-trisphosphate receptor: role in Ca(2+) signaling and disease

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Apoptosis regulation by Bcl-x(L) modulation of mammalian inositol 1,4,5-trisphosphate receptor channel isoform gating.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Structural studies of inositol 1,4,5-trisphosphate receptor: coupling ligand binding to channel gating

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Selective regulation of IP3-receptor-mediated Ca2+ signaling and apoptosis by the BH4 domain of Bcl-2 versus Bcl-Xl.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Targeting Bcl-2 based on the interaction of its BH4 domain with the inositol 1,4,5-trisphosphate receptor.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Induction of Ca²+-driven apoptosis in chronic lymphocytic leukemia cells by peptide-mediated disruption of Bcl-2-IP3 receptor interaction

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

The apoptotic regulator Nrz controls cytoskeletal dynamics via the regulation of Ca2+ trafficking in the zebrafish blastula

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Gastrulation in zebrafish -- all just about adhesion?

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

The zebrafish bcl-2 homologue Nrz controls development during somitogenesis and gastrulation via apoptosis-dependent and -independent mechanisms.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Cytoskeleton dynamics in early zebrafish development: A matter of phosphorylation?

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Calcium and the damage pathways in muscular dystrophy

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Bcl-2 overexpression prevents calcium overload and subsequent apoptosis in dystrophic myotubes

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Calcium homeostasis in vascular smooth muscle cells is altered in type 2 diabetes by Bcl-2 protein modulation of InsP3R calcium release channels

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

The Bcl-2 gene polymorphism rs956572AA increases inositol 1,4,5-trisphosphate receptor-mediated endoplasmic reticulum calcium release in subjects with bipolar disorder

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Bcl-2 SNP rs956572 associates with disrupted intracellular calcium homeostasis in bipolar I disorder

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Bcl-2 polymorphism influences gray matter volume in the ventral striatum in healthy humans

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

The mesolimbic dopamine reward circuit in depression

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Initial testing (stage 1) of the BH3 mimetic ABT-263 by the pediatric preclinical testing program

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Bcl-2 inhibitors: small molecules with a big impact on cancer therapy

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Non-peptidic small molecule inhibitors against Bcl-2 for cancer therapy.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

The Bcl-2 homology domain 3 mimetic ABT-737 targets the apoptotic machinery in acute lymphoblastic leukemia resulting in synergistic in vitro and in vivo interactions with established drugs

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

The Bcl-2 inhibitor ABT-263 enhances the response of multiple chemotherapeutic regimens in hematologic tumors in vivo

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

BCL2/BCL-X(L) inhibition induces apoptosis, disrupts cellular calcium homeostasis, and prevents platelet activation

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Bcl-xL-inhibitory BH3 mimetics can induce a transient thrombocytopathy that undermines the hemostatic function of platelets

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Programmed anuclear cell death delimits platelet life span.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Distinct roles of mitochondria- and ER-localized Bcl-xL in apoptosis resistance and Ca2+ homeostasis

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

BCL-2 and BCL-XL restrict lineage choice during hematopoietic differentiation

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Molecular basis of the interaction between the antiapoptotic Bcl-2 family proteins and the proapoptotic protein ASPP2

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

BH4 domain of antiapoptotic Bcl-2 family members closes voltage-dependent anion channel and inhibits apoptotic mitochondrial changes and cell death

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

The voltage-dependent anion channel (VDAC): function in intracellular signalling, cell life and cell death.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

VDAC, a multi-functional mitochondrial protein regulating cell life and death

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

The voltage-dependent anion channel-1 modulates apoptotic cell death.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

The expression level of the voltage-dependent anion channel controls life and death of the cell

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Bcl-x(L) blocks a mitochondrial inner membrane channel and prevents Ca2+ overload-mediated cell death

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Oligomerization of the mitochondrial protein voltage-dependent anion channel is coupled to the induction of apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Hexokinase-I protection against apoptotic cell death is mediated via interaction with the voltage-dependent anion channel-1: mapping the site of binding.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

The VDAC1 N-terminus is essential both for apoptosis and the protective effect of anti-apoptotic proteins.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Structure-based analysis of VDAC1: N-terminus location, translocation, channel gating and association with anti-apoptotic proteins.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

NMR structural investigation of the mitochondrial outer membrane protein VDAC and its interaction with antiapoptotic Bcl-xL.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Bax inhibitor-1, a mammalian apoptosis suppressor identified by functional screening in yeast

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

BI-1 regulates an apoptosis pathway linked to endoplasmic reticulum stress

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Cardiolipin, phosphatidylserine, and BH4 domain of Bcl-2 family regulate Ca2+/H+ antiporter activity of human Bax inhibitor-1.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

BI-1 protects cells from oxygen glucose deprivation by reducing the calcium content of the endoplasmic reticulum

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

The ancient cell death suppressor BAX inhibitor-1.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Human Golgi antiapoptotic protein modulates intracellular calcium fluxes

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

TMBIM3/GRINA is a novel unfolded protein response (UPR) target gene that controls apoptosis through the modulation of ER calcium homeostasis

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

ASPP: a new family of oncogenes and tumour suppressor genes

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

ASPP2: a gene that controls life and death in vivo

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

The ASPP interaction network: electrostatic differentiation between pro- and anti-apoptotic proteins

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

New insights into the expanding complexity of the tumor suppressor ASPP2.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Insight into the structural basis of pro- and antiapoptotic p53 modulation by ASPP proteins

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

A model for the interaction between NF-kappa-B and ASPP2 suggests an I-kappa-B-like binding mechanism

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

The structure and interactions of the proline-rich domain of ASPP2

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Insights into the structure and protein-protein interactions of the pro-apoptotic protein ASPP2.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

DeathBase: a database on structure, evolution and function of proteins involved in apoptosis and other forms of cell death

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-012-1118-Y

Ca(2+) signaling mechanisms of cell survival and cell death: an introduction

21 January 2018

1 reference

12 December 2020

Apoptosis induced by withdrawal of interleukin-3 (IL-3) from an IL-3-dependent hematopoietic cell line is associated with repartitioning of intracellular calcium and is blocked by enforced Bcl-2 oncoprotein production

1 reference

12 December 2020

Endoplasmic reticulum/mitochondria calcium cross-talk.

1 reference

12 December 2020

Mitochondrial calcium signalling: message of life and death

1 reference

12 December 2020

Regulation of autophagy by the inositol trisphosphate receptor

1 reference

12 December 2020

Hierarchical regulation of mitochondrion-dependent apoptosis by BCL-2 subfamilies

1 reference

12 December 2020

Suppression of IP3-mediated calcium release and apoptosis by Bcl-2 involves the participation of protein phosphatase 1

1 reference

12 December 2020

10.1007/S00018-012-1118-Y

Identifiers

DOI

1 reference

27 August 2017

1 reference