(Q38626688)

4 September 2017

Inflammatory and fibrotic responses of cardiac fibroblasts to myocardial damage associated molecular patterns (DAMPs). (English)

1 reference

4 September 2017

0 references

1 reference

Neil A Turner

1

0 references

publication date

2 November 2015

1 reference

full work available at URL

4 September 2017

12

1 reference

based on heuristic

inferred from page(s)

http://eprints.whiterose.ac.uk/99183/

content deliverer

White Rose Research Online

copyright license

Creative Commons Attribution-NonCommercial-NoDerivs 4.0 International

0 references

published in

Journal of Molecular and Cellular Cardiology

1 reference

4 September 2017

94

1 reference

4 September 2017

189-200

1 reference

Cardiac fibroblasts: at the heart of myocardial remodeling

4 September 2017

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

The cardiac fibroblast: therapeutic target in myocardial remodeling and failure

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Cardiac fibroblast: the renaissance cell

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Myocardial remodeling after infarction: the role of myofibroblasts

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

The origin of fibroblasts and mechanism of cardiac fibrosis

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Function and fate of myofibroblasts after myocardial infarction

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Regulation of myocardial matrix metalloproteinase expression and activity by cardiac fibroblasts

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Cardiac intercellular communication: are myocytes and fibroblasts fair-weather friends?

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Crosstalk between fibroblasts and inflammatory cells

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

The role of cardiac fibroblasts in the transition from inflammation to fibrosis following myocardial infarction

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Hypoxia induces apoptosis with enhanced expression of Fas antigen messenger RNA in cultured neonatal rat cardiomyocytes

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Differential vulnerability to oxidative stress in rat cardiac myocytes versus fibroblasts.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Innate immune signaling in cardiac ischemia

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

The emerging role of innate immunity in the heart and vascular system: for whom the cell tolls

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

The inflammatory response in myocardial infarction

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

The immune system and cardiac repair.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Monocyte and macrophage contributions to cardiac remodeling.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Role of inflammatory cells in fibroblast activation

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

DAMPening inflammation by modulating TLR signalling

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Danger signals in the initiation of the inflammatory response after myocardial infarction

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Inflammation and metabolic disorders

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Innate immunity and cardiomyocytes in ischemic heart disease

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Crosstalk between the renin-angiotensin system and the advance glycation end product axis in the heart: role of the cardiac fibroblast

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Molecular mechanisms of AGE/RAGE-mediated fibrosis in the diabetic heart

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Necrotic myocardial cells release damage-associated molecular patterns that provoke fibroblast activation in vitro and trigger myocardial inflammation and fibrosis in vivo.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

NOD1 activation induces cardiac dysfunction and modulates cardiac fibrosis and cardiomyocyte apoptosis

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Mechanisms of disease: Toll-like receptors in cardiovascular disease

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Toll-like receptor signaling: a critical modulator of cell survival and ischemic injury in the heart

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Expanding TRAF function: TRAF3 as a tri-faced immune regulator

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

NOD-like receptors: versatile cytosolic sentinels.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Role of the inflammasome in myocardial infarction

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Cellular signalling of the receptor for advanced glycation end products (RAGE).

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Myocardial ischemia/reperfusion injury is mediated by leukocytic toll-like receptor-2 and reduced by systemic administration of a novel anti-toll-like receptor-2 antibody

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Toll-like receptors 2-deficient mice are protected against postischemic coronary endothelial dysfunction

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Left ventricular dilation in toll-like receptor 2 deficient mice after myocardial ischemia/reperfusion through defective scar formation

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Akt or phosphoinositide-3-kinase inhibition reverses cardio-protection in Toll-like receptor 2 deficient mice

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Toll-like receptor-2 modulates ventricular remodeling after myocardial infarction.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Inhibition of Toll-like receptor 2 reduces cardiac fibrosis by attenuating macrophage-mediated inflammation.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Role of extracellular RNA and TLR3-Trif signaling in myocardial ischemia-reperfusion injury

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Toll-like receptor 4 stimulation initiates an inflammatory response that decreases cardiomyocyte contractility

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Toll-like receptor 4 mediates ischemia/reperfusion injury of the heart

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

High mobility group [corrected] box 1 mediates neutrophil recruitment in myocardial ischemia-reperfusion injury through toll like receptor 4-related pathway

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

The HMGB1-TLR4 axis contributes to myocardial ischemia/reperfusion injury via regulation of cardiomyocyte apoptosis

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Toll-like receptor 4 knockout protects against isoproterenol-induced cardiac fibrosis: the role of autophagy.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Cardiomyocyte Toll-like receptor 4 is involved in heart dysfunction following septic shock or myocardial ischemia

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Reduced cardiac hypertrophy in toll-like receptor 4-deficient mice following pressure overload

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Protection against myocardial ischemia/reperfusion injury in TLR4-deficient mice is mediated through a phosphoinositide 3-kinase-dependent mechanism

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Toll-like receptor 4 deficiency: smaller infarcts, but no gain in function

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Myocardial ischemia activates an injurious innate immune signaling via cardiac heat shock protein 60 and Toll-like receptor 4.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Angiotensin Ⅱ Activates MCP-1 and Induces Cardiac Hypertrophy and Dysfunction via Toll-like Receptor 4.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Reduced myocardial ischemia-reperfusion injury in toll-like receptor 4-deficient mice

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Toll-like receptor-4 modulates survival by induction of left ventricular remodeling after myocardial infarction in mice.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Toll-like receptor 4 modulates myocardial ischaemia-reperfusion injury: Role of matrix metalloproteinases

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Toll-Like Receptor 4 Mediates Maladaptive Left Ventricular Remodeling and Impairs Cardiac Function After Myocardial Infarction

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Toll-like receptor-2 mediates adaptive cardiac hypertrophy in response to pressure overload through interleukin-1β upregulation via nuclear factor κB activation

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Toll-like receptor 5 deficiency exacerbates cardiac injury and inflammation induced by myocardial ischaemia-reperfusion in the mouse.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Alterations in the interleukin-1/interleukin-1 receptor antagonist balance modulate cardiac remodeling following myocardial infarction in the mouse

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Interleukin-1 receptor type I signaling critically regulates infarct healing and cardiac remodeling.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

IL-1 induces proinflammatory leukocyte infiltration and regulates fibroblast phenotype in the infarcted myocardium.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

NOD2 deficiency protects against cardiac remodeling after myocardial infarction in mice.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

NOD2 deletion promotes cardiac hypertrophy and fibrosis induced by pressure overload

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

RAGE modulates myocardial injury consequent to LAD infarction via impact on JNK and STAT signaling in a murine model

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

High-Mobility Group Box-1 in Ischemia-Reperfusion Injury of the Heart

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Receptor for advanced glycation end products (RAGE) and implications for the pathophysiology of heart failure

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Cutting edge: IL-1α is a crucial danger signal triggering acute myocardial inflammation during myocardial infarction

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

S100A1 is released from ischemic cardiomyocytes and signals myocardial damage via Toll-like receptor 4

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Therapeutic targeting of innate immunity in the failing heart

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Tissue-specific mRNA expression profiles of human toll-like receptors and related genes

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Toll-like receptor stimulation in cardiomyoctes decreases contractility and initiates an NF-kappaB dependent inflammatory response

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Role of TLR-2 in the activation of nuclear factor kappaB by oxidative stress in cardiac myocytes

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

The NLRP3 inflammasome is up-regulated in cardiac fibroblasts and mediates myocardial ischaemia-reperfusion injury.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Protein kinase Cε-calcineurin cosignaling downstream of toll-like receptor 4 downregulates fibrosis and induces wound healing gene expression in cardiac myofibroblasts.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Toll-like receptor 9 mediated responses in cardiac fibroblasts

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Mitochondrial NLRP3 protein induces reactive oxygen species to promote Smad protein signaling and fibrosis independent from the inflammasome.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

HMGB1-stimulated human primary cardiac fibroblasts exert a paracrine action on human and murine cardiac stem cells

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Cardiac fibroblast-dependent extracellular matrix accumulation is associated with diastolic stiffness in type 2 diabetes

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

S100a8/a9 released by CD11b+Gr1+ neutrophils activates cardiac fibroblasts to initiate angiotensin II-Induced cardiac inflammation and injury.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

S100 proteins in mouse and man: from evolution to function and pathology (including an update of the nomenclature)

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

S100 proteins expressed in phagocytes: a novel group of damage-associated molecular pattern molecules

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Immunohistochemical detection of S100A1 in the postmortem diagnosis of acute myocardial infarction

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Proinflammatory activities of S100: proteins S100A8, S100A9, and S100A8/A9 induce neutrophil chemotaxis and adhesion

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

The role of interleukin-1 in the failing heart

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

The role of IL-1 in the pathogenesis of heart disease.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Identification of a key pathway required for the sterile inflammatory response triggered by dying cells

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Effects of interleukin-1 on cardiac fibroblast function: relevance to post-myocardial infarction remodelling

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Endotoxin-activated cultured neonatal rat cardiomyocytes express functional surface-associated interleukin-1alpha

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Mechanism of TNFalpha-induced IL-1alpha, IL-1beta and IL-6 expression in human cardiac fibroblasts: effects of statins and thiazolidinediones.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Interleukin-1alpha stimulates proinflammatory cytokine expression in human cardiac myofibroblasts.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Human cardiac fibroblasts express ICAM-1, E-selectin and CXC chemokines in response to proinflammatory cytokine stimulation

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

p38 MAPK alpha mediates cytokine-induced IL-6 and MMP-3 expression in human cardiac fibroblasts

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Interleukin-1 has opposing effects on connective tissue growth factor and tenascin-C expression in human cardiac fibroblasts.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Combined effects of interleukin-1α and transforming growth factor-β1 on modulation of human cardiac fibroblast function

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Modulatory effect of interleukin-1α on expression of structural matrix proteins, MMPs and TIMPs in human cardiac myofibroblasts: role of p38 MAP kinase

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Peroxisome proliferator-activated receptor gamma-independent effects of thiazolidinediones on human cardiac myofibroblast function

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

IL-1β suppresses TGF-β-mediated myofibroblast differentiation in cardiac fibroblasts.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Cytokines regulate matrix metalloproteinases and migration in cardiac fibroblasts

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

IL-1 signaling in atherosclerosis: sibling rivalry

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Targeting interleukin-1 in heart failure and inflammatory heart disease

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

HMGB1 and RAGE in inflammation and cancer

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Inhibition of high-mobility group box 1 improves myocardial fibrosis and dysfunction in diabetic cardiomyopathy.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Up-regulated HMGB1 in EAM directly led to collagen deposition by a PKCβ/Erk1/2-dependent pathway: cardiac fibroblast/myofibroblast might be another source of HMGB1

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

HMGB1 attenuates cardiac remodelling in the failing heart via enhanced cardiac regeneration and miR-206-mediated inhibition of TIMP-3

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Exogenous high-mobility group box 1 protein prevents postinfarction adverse myocardial remodeling through TGF-β/Smad signaling pathway

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Extracellular heat shock protein 60, cardiac myocytes, and apoptosis

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Elevated circulating levels of heat shock protein 70 are related to systemic inflammatory reaction through monocyte Toll signal in patients with heart failure after acute myocardial infarction

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Human myocardium releases heat shock protein 27 (HSP27) after global ischemia: the proinflammatory effect of extracellular HSP27 through toll-like receptor (TLR)-2 and TLR4

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Lack of Fibronectin-EDA Promotes Survival and Prevents Adverse Remodeling and Heart Function Deterioration After Myocardial Infarction

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Myofibroblasts and mechano-regulation of connective tissue remodelling

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Atherosclerotic lesion development and Toll like receptor 2 and 4 responsiveness

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

The extra domain A of fibronectin activates Toll-like receptor 4.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

FibronectinEDA promotes chronic cutaneous fibrosis through Toll-like receptor signaling.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Tenascin-C modulates adhesion of cardiomyocytes to extracellular matrix during tissue remodeling after myocardial infarction.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

The role of tenascin C in cardiovascular disease.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Tenascin-C: Its functions as an integrin ligand

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Tenascin-C is an endogenous activator of Toll-like receptor 4 that is essential for maintaining inflammation in arthritic joint disease.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Tenascin-C as predictor of left ventricular remodeling and mortality in patients with dilated cardiomyopathy.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Serum tenascin-C might be a novel predictor of left ventricular remodeling and prognosis after acute myocardial infarction

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Tenascin-C may aggravate left ventricular remodeling and function after myocardial infarction in mice.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Tenascin-C may accelerate cardiac fibrosis by activating macrophages via the integrin αVβ3/nuclear factor-κB/interleukin-6 axis.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Tenascin-C regulates recruitment of myofibroblasts during tissue repair after myocardial injury.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Clinical diabetic cardiomyopathy: a two-faced disease with restrictive and dilated phenotypes

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Diabetes-associated cardiac fibrosis: Cellular effectors, molecular mechanisms and therapeutic opportunities

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Advanced glycation end products: role in pathology of diabetic cardiomyopathy

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Type II diabetes promotes a myofibroblast phenotype in cardiac fibroblasts

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Investigating inherent functional differences between human cardiac fibroblasts cultured from nondiabetic and Type 2 diabetic donors.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Inflammasome activation of cardiac fibroblasts is essential for myocardial ischemia/reperfusion injury

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

The inflammasome promotes adverse cardiac remodeling following acute myocardial infarction in the mouse.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Activation in M1 but not M2 Macrophages Contributes to Cardiac Remodeling after Myocardial Infarction in Rats: a Critical Role of the Calcium Sensing Receptor/NRLP3 Inflammasome

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Nlrp3 plays no role in acute cardiac infarction due to low cardiac expression.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

A novel pharmacologic inhibitor of the NLRP3 inflammasome limits myocardial injury after ischemia-reperfusion in the mouse

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Pharmacologic Inhibition of the NLRP3 Inflammasome Preserves Cardiac Function After Ischemic and Nonischemic Injury in the Mouse

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Lack of specificity of fibroblast-specific protein 1 in cardiac remodeling and fibrosis

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Origin of cardiac fibroblasts and the role of periostin

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Cardiac fibroblasts are essential for the adaptive response of the murine heart to pressure overload

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

A potent far-upstream enhancer in the mouse pro alpha 2(I) collagen gene regulates expression of reporter genes in transgenic mice

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Fibroblast-specific Expression of a Kinase-deficient Type II Transforming Growth Factor β (TGFβ) Receptor Leads to Paradoxical Activation of TGFβ Signaling Pathways with Fibrosis in Transgenic Mice

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Ligand-dependent genetic recombination in fibroblasts : a potentially powerful technique for investigating gene function in fibrosis

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Inducible lineage-specific deletion of TbetaRII in fibroblasts defines a pivotal regulatory role during adult skin wound healing.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Cardiac fibroblast glycogen synthase kinase-3β regulates ventricular remodeling and dysfunction in ischemic heart

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Mesenchymal-endothelial transition contributes to cardiac neovascularization.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

MicroRNAs: target recognition and regulatory functions

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

MicroRNA in TLR signaling and endotoxin tolerance.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

MicroRNA-146a: A Dominant, Negative Regulator of the Innate Immune Response

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Increased expression of microRNA-146a decreases myocardial ischaemia/reperfusion injury

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Treating inflammation by blocking interleukin-1 in a broad spectrum of diseases

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Effects of interleukin-1 blockade with anakinra on adverse cardiac remodeling and heart failure after acute myocardial infarction [from the Virginia Commonwealth University-Anakinra Remodeling Trial (2) (VCU-ART2) pilot study].

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

The effect of interleukin-1 receptor antagonist therapy on markers of inflammation in non-ST elevation acute coronary syndromes: the MRC-ILA Heart Study

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

Interleukin-1 blockade with anakinra to prevent adverse cardiac remodeling after acute myocardial infarction (Virginia Commonwealth University Anakinra Remodeling Trial [VCU-ART] Pilot study).

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2015.11.002

10.1016/J.YJMCC.2015.11.002

7 January 2021

Identifiers

DOI

1 reference

10.1016/J.YJMCC.2015.11.002

4 September 2017

0 references

Dimensions Publication ID

1012100435

0 references

Microsoft Academic ID

0 references

1 reference