(Q28274739)

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

The NLRP3 inflammasome in health and disease: the good, the bad and the ugly

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

The diverse role of RIP kinases in necroptosis and inflammation

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

The inflammasome: a molecular platform triggering activation of inflammatory caspases and processing of proIL-beta

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Phosphorylation of the adaptor ASC acts as a molecular switch that controls the formation of speck-like aggregates and inflammasome activity

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Unified polymerization mechanism for the assembly of ASC-dependent inflammasomes

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Prion-like polymerization underlies signal transduction in antiviral immune defense and inflammasome activation

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Gain-of-function Pyrin mutations induce NLRP3 protein-independent interleukin-1β activation and severe autoinflammation in mice

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

NLRP1 inflammasome activation induces pyroptosis of hematopoietic progenitor cells

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Cryopyrinopathies: update on pathogenesis and treatment.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

An activating NLRC4 inflammasome mutation causes autoinflammation with recurrent macrophage activation syndrome

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Mutation of NLRC4 causes a syndrome of enterocolitis and autoinflammation

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

K⁺ efflux is the common trigger of NLRP3 inflammasome activation by bacterial toxins and particulate matter

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Mitochondrial apoptosis is dispensable for NLRP3 inflammasome activation but non-apoptotic caspase-8 is required for inflammasome priming

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Critical functions of priming and lysosomal damage for NLRP3 activation

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Cryopyrin activates the inflammasome in response to toxins and ATP

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

A role for mitochondria in NLRP3 inflammasome activation

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Autophagy proteins regulate innate immune responses by inhibiting the release of mitochondrial DNA mediated by the NALP3 inflammasome

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Oxidized mitochondrial DNA activates the NLRP3 inflammasome during apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Mitochondrial cardiolipin is required for Nlrp3 inflammasome activation

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

The calcium-sensing receptor regulates the NLRP3 inflammasome through Ca2+ and cAMP.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Gout-associated uric acid crystals activate the NALP3 inflammasome

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Activation of the NLRP3 inflammasome by islet amyloid polypeptide provides a mechanism for enhanced IL-1β in type 2 diabetes

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

The NLRP3 inflammasome instigates obesity-induced inflammation and insulin resistance

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Fatty acid-induced NLRP3-ASC inflammasome activation interferes with insulin signaling

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

The NALP3 inflammasome is involved in the innate immune response to amyloid-beta

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

NLRP3 is activated in Alzheimer's disease and contributes to pathology in APP/PS1 mice

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

The Salmonella invasin SipB induces macrophage apoptosis by binding to caspase-1

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Shigella-induced apoptosis is dependent on caspase-1 which binds to IpaB

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Pyroptotic death storms and cytopenia

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Pyroptotic cell death defends against intracellular pathogens

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Cell death by pyroptosis drives CD4 T-cell depletion in HIV-1 infection

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Divergence of IL-1, IL-18, and cell death in NLRP3 inflammasomopathies

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Active caspase-1 is a regulator of unconventional protein secretion

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Rapid induction of inflammatory lipid mediators by the inflammasome in vivo

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Caspase-11 protects against bacteria that escape the vacuole

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Cytoplasmic LPS activates caspase-11: implications in TLR4-independent endotoxic shock

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Caspase-11 activates a canonical NLRP3 inflammasome by promoting K(+) efflux

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

NLRP3 inflammasome activation downstream of cytoplasmic LPS recognition by both caspase-4 and caspase-5.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Non-canonical inflammasome activation targets caspase-11

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Gasdermin D is an executor of pyroptosis and required for interleukin-1β secretion

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Cleavage of GSDMD by inflammatory caspases determines pyroptotic cell death

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Caspase-11 cleaves gasdermin D for non-canonical inflammasome signalling.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Caspase-11 Requires the Pannexin-1 Channel and the Purinergic P2X7 Pore to Mediate Pyroptosis and Endotoxic Shock

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Mechanisms of unconventional secretion of IL-1 family cytokines

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Autophagy-based unconventional secretory pathway for extracellular delivery of IL-1β

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Loss of the autophagy protein Atg16L1 enhances endotoxin-induced IL-1beta production

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Activation of autophagy by inflammatory signals limits IL-1β production by targeting ubiquitinated inflammasomes for destruction

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Single-cell imaging of caspase-1 dynamics reveals an all-or-none inflammasome signaling response.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Real-time single-cell imaging of protein secretion

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

In vivo imaging of inflammasome activation reveals a subcapsular macrophage burst response that mobilizes innate and adaptive immunity

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

The neutrophil NLRC4 inflammasome selectively promotes IL-1β maturation without pyroptosis during acute Salmonella challenge

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Neutrophil IL-1β processing induced by pneumolysin is mediated by the NLRP3/ASC inflammasome and caspase-1 activation and is dependent on K+ efflux

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Inflammasome activators induce interleukin-1α secretion via distinct pathways with differential requirement for the protease function of caspase-1.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Caspase-1-dependent processing of pro-interleukin-1beta is cytosolic and precedes cell death

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Pore formation triggered by Legionella spp. is an Nlrc4 inflammasome-dependent host cell response that precedes pyroptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Caspase-1-dependent pore formation during pyroptosis leads to osmotic lysis of infected host macrophages

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Inhibitory effects of chloride on the activation of caspase-1, IL-1beta secretion, and cytolysis by the P2X7 receptor

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

A20 restricts ubiquitination of pro-interleukin-1β protein complexes and suppresses NLRP3 inflammasome activity

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Caspase-8 blocks kinase RIPK3-mediated activation of the NLRP3 inflammasome.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

The regulation of TNF signalling: what a tangled web we weave

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Stimulation of Toll-like receptor 3 and 4 induces interleukin-1beta maturation by caspase-8.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Inhibitor of apoptosis proteins limit RIP3 kinase-dependent interleukin-1 activation.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Inhibition of Histone Deacetylases Permits Lipopolysaccharide-Mediated Secretion of Bioactive IL-1β via a Caspase-1-Independent Mechanism.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Cutting edge: Endoplasmic reticulum stress licenses macrophages to produce mature IL-1β in response to TLR4 stimulation through a caspase-8- and TRIF-dependent pathway

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Proapoptotic chemotherapeutic drugs induce noncanonical processing and release of IL-1β via caspase-8 in dendritic cells

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

XIAP restricts TNF- and RIP3-dependent cell death and inflammasome activation

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Participation of c-FLIP in NLRP3 and AIM2 inflammasome activation.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

RIPK3 promotes cell death and NLRP3 inflammasome activation in the absence of MLKL

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

A TNFSF15 disease-risk polymorphism increases pattern-recognition receptor-induced signaling through caspase-8-induced IL-1.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

A RIPK3-caspase 8 complex mediates atypical pro-IL-1β processing

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Dectin-1 is an extracellular pathogen sensor for the induction and processing of IL-1β via a noncanonical caspase-8 inflammasome

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Caspase-8 modulates dectin-1 and complement receptor 3-driven IL-1β production in response to β-glucans and the fungal pathogen, Candida albicans

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Internalized Cryptococcus neoformans Activates the Canonical Caspase-1 and the Noncanonical Caspase-8 Inflammasomes

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Catalytic activity of the caspase-8-FLIP(L) complex inhibits RIPK3-dependent necrosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

RIP3 mediates the embryonic lethality of caspase-8-deficient mice

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

New insights into the regulation of innate immunity by caspase-8.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

More to life than death: molecular determinants of necroptotic and non-necroptotic RIP3 kinase signaling

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Salmonella infection induces recruitment of Caspase-8 to the inflammasome to modulate IL-1β production

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

FADD and caspase-8 mediate priming and activation of the canonical and noncanonical Nlrp3 inflammasomes.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Cutting edge: FAS (CD95) mediates noncanonical IL-1β and IL-18 maturation via caspase-8 in an RIP3-independent manner

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

ASC controls IFN-γ levels in an IL-18-dependent manner in caspase-1-deficient mice infected with Francisella novicida

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

AIM2/ASC triggers caspase-8-dependent apoptosis in Francisella-infected caspase-1-deficient macrophages.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Dietary modulation of the microbiome affects autoinflammatory disease

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

A Role for Gut Microbiota and the Metabolite-Sensing Receptor GPR43 in a Murine Model of Gout

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Metabolite-sensing receptors GPR43 and GPR109A facilitate dietary fibre-induced gut homeostasis through regulation of the inflammasome.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

NLRP6 inflammasome regulates colonic microbial ecology and risk for colitis

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Microbiota-Modulated Metabolites Shape the Intestinal Microenvironment by Regulating NLRP6 Inflammasome Signaling.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Conditional deletion of caspase-8 in macrophages alters macrophage activation in a RIPK-dependent manner.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Microbiota regulates immune defense against respiratory tract influenza A virus infection

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Doxorubicin and daunorubicin induce processing and release of interleukin-1β through activation of the NLRP3 inflammasome

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

RIP3 induces apoptosis independent of pronecrotic kinase activity

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Activity of protein kinase RIPK3 determines whether cells die by necroptosis or apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Caspase-8 scaffolding function and MLKL regulate NLRP3 inflammasome activation downstream of TLR3

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

ASC is an activating adaptor for NF-kappa B and caspase-8-dependent apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

ASC, a novel 22-kDa protein, aggregates during apoptosis of human promyelocytic leukemia HL-60 cells

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

AIM2 and NLRP3 inflammasomes activate both apoptotic and pyroptotic death pathways via ASC.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

The Inflammasome Adaptor ASC Induces Procaspase-8 Death Effector Domain Filaments

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Caspase-8 as an Effector and Regulator of NLRP3 Inflammasome Signaling

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Acute lipopolysaccharide priming boosts inflammasome activation independently of inflammasome sensor induction

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Cutting edge: TLR signaling licenses IRAK1 for rapid activation of the NLRP3 inflammasome

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Inflammasome priming by lipopolysaccharide is dependent upon ERK signaling and proteasome function

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Non-transcriptional priming and deubiquitination regulate NLRP3 inflammasome activation

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Linear ubiquitination prevents inflammation and regulates immune signalling

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Recruitment of the linear ubiquitin chain assembly complex stabilizes the TNF-R1 signaling complex and is required for TNF-mediated gene induction

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Sustained elevation of serum interleukin-18 and its association with hemophagocytic lymphohistiocytosis in XIAP deficiency

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Oversecretion of IL-18 in haemophagocytic lymphohistiocytosis: a novel marker of disease activity

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Immunodeficiency, autoinflammation and amylopectinosis in humans with inherited HOIL-1 and LUBAC deficiency

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Human HOIP and LUBAC deficiency underlies autoinflammation, immunodeficiency, amylopectinosis, and lymphangiectasia

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Inhibition of NF-κB Signaling Retards Eosinophilic Dermatitis in SHARPIN-Deficient Mice

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

SHARPIN forms a linear ubiquitin ligase complex regulating NF-κB activity and apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Cutting Edge: RIP1 kinase activity is dispensable for normal development but is a key regulator of inflammation in SHARPIN-deficient mice.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

The Inflammatory Caspases-1 and -11 Mediate the Pathogenesis of Dermatitis in Sharpin-Deficient Mice.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Linear ubiquitination-mediated NF-κB regulation and its related disorders

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Cutting edge: SHARPIN is required for optimal NLRP3 inflammasome activation

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Systems analysis identifies an essential role for SHANK-associated RH domain-interacting protein (SHARPIN) in macrophage Toll-like receptor 2 (TLR2) responses

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

The linear ubiquitin assembly complex (LUBAC) is essential for NLRP3 inflammasome activation.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

The ubiquitin-modifying enzyme A20 restricts ubiquitination of the kinase RIPK3 and protects cells from necroptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Loss-of-function mutations in TNFAIP3 leading to A20 haploinsufficiency cause an early-onset autoinflammatory disease

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Apoptotic caspases suppress mtDNA-induced STING-mediated type I IFN production

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Apoptotic caspases prevent the induction of type I interferons by mitochondrial DNA.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

A perspective on mammalian caspases as positive and negative regulators of inflammation

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

cFLIP regulates skin homeostasis and protects against TNF-induced keratinocyte apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

MAVS Promotes Inflammasome Activation by Targeting ASC for K63-Linked Ubiquitination via the E3 Ligase TRAF3.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Cytosolic double-stranded RNA activates the NLRP3 inflammasome via MAVS-induced membrane permeabilization and K+ efflux

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

The adaptor MAVS promotes NLRP3 mitochondrial localization and inflammasome activation

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Mitochondrial protein mitofusin 2 is required for NLRP3 inflammasome activation after RNA virus infection

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

The Mitochondrial Phosphatase PGAM5 Is Dispensable for Necroptosis but Promotes Inflammasome Activation in Macrophages.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Inflammasome activation leads to Caspase-1-dependent mitochondrial damage and block of mitophagy

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Post-translational control of RIPK3 and MLKL mediated necroptotic cell death

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Ars Moriendi; the art of dying well - new insights into the molecular pathways of necroptotic cell death

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Toxin-induced necroptosis is a major mechanism of Staphylococcus aureus lung damage.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Activation of necroptosis in multiple sclerosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

A role of RIP3-mediated macrophage necrosis in atherosclerosis development.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

The role of interferon-β in the treatment of multiple sclerosis and experimental autoimmune encephalomyelitis - in the perspective of inflammasomes

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

RNA viruses promote activation of the NLRP3 inflammasome through a RIP1-RIP3-DRP1 signaling pathway.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Inflammasome-independent IL-1β mediates autoinflammatory disease in Pstpip2-deficient mice

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Critical role for inflammasome-independent IL-1β production in osteomyelitis

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Caspase-8 and RIP kinases regulate bacteria-induced innate immune responses and cell death.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Caspase-8 promotes NLRP1/NLRP3 inflammasome activation and IL-1β production in acute glaucoma

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

RIPK1 regulates RIPK3-MLKL-driven systemic inflammation and emergency hematopoiesis.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00018-016-2205-2

Inflammasome-mediated dysbiosis regulates progression of NAFLD and obesity

21 January 2018

1 reference

12 December 2020

ER stress activates the NLRP3 inflammasome via an UPR-independent pathway

1 reference

12 December 2020

A bacterial invasin induces macrophage apoptosis by binding directly to ICE

1 reference

12 December 2020

Inflammatory caspases are innate immune receptors for intracellular LPS.

1 reference

12 December 2020

Noncanonical inflammasome activation by intracellular LPS independent of TLR4.

1 reference

12 December 2020

NLRP3 inflammasome and its inhibitors: a review

1 reference

12 December 2020

Negative regulation of the NLRP3 inflammasome by A20 protects against arthritis

1 reference

12 December 2020

Caspase-8 mediates caspase-1 processing and innate immune defense in response to bacterial blockade of NF-κB and MAPK signaling

1 reference

12 December 2020

Sharpin prevents skin inflammation by inhibiting TNFR1-induced keratinocyte apoptosis

1 reference

12 December 2020

Caspase-1-independent, Fas/Fas ligand-mediated IL-18 secretion from macrophages causes acute liver injury in mice

1 reference

12 December 2020

Translocation of interleukin-1β into a vesicle intermediate in autophagy-mediated secretion

1 reference

12 December 2020

TRAF2 regulates TNF and NF-κB signalling to suppress apoptosis and skin inflammation independently of Sphingosine kinase 1

1 reference

12 December 2020

NLRP3 inflammasomes are required for atherogenesis and activated by cholesterol crystals

1 reference

12 December 2020

SAP and XIAP deficiency in hemophagocytic lymphohistiocytosis

1 reference

12 December 2020

TNFR1-dependent cell death drives inflammation in Sharpin-deficient mice.

1 reference

12 December 2020

Inhibitor of apoptosis (IAP) proteins-modulators of cell death and inflammation

1 reference

12 December 2020

Diverse Activators of the NLRP3 Inflammasome Promote IL-1β Secretion by Triggering Necrosis

1 reference

12 December 2020

IL-1 signaling in atherosclerosis: sibling rivalry

1 reference

12 December 2020

Ambiguities in NLRP3 inflammasome regulation: is there a role for mitochondria?

1 reference

12 December 2020

10.1007/S00018-016-2205-2

Identifiers

DOI

1 reference

Consolidated OpenCitations Corpus – April 2017

1 reference

Consolidated OpenCitations Corpus – April 2017

PubMed publication ID

27066895

1 reference

Consolidated OpenCitations Corpus – April 2017