(Q38071479)

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The mitochondrial H(+)-ATP synthase and the lipogenic switch: new core components of metabolic reprogramming in induced pluripotent stem (iPS) cells.

scientific article published on 15 January 2012

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21 February 2020

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The mitochondrial H(+)-ATP synthase and the lipogenic switch: new core components of metabolic reprogramming in induced pluripotent stem (iPS) cells (English)

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21 February 2020

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21 February 2020

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Alejandro Vazquez-Martin

1

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21 February 2020

Bruna Corominas-Faja

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21 February 2020

Sílvia Cufi

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21 February 2020

Luciano Vellon

4

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21 February 2020

Cristina Oliveras-Ferraros

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21 February 2020

Octavio J Menendez

6

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21 February 2020

Ruth Lupu

8

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21 February 2020

Javier A Menendez

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21 February 2020

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15 January 2012

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21 February 2020

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21 February 2020

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21 February 2020

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21 February 2020

207-218

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21 February 2020

Metformin limits the tumourigenicity of iPS cells without affecting their pluripotency

1 reference

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1 reference

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1 reference

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The metabolome of induced pluripotent stem cells reveals metabolic changes occurring in somatic cell reprogramming

1 reference

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AMP-activated protein kinase: an energy sensor that regulates all aspects of cell function

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Somatic oxidative bioenergetics transitions into pluripotency-dependent glycolysis to facilitate nuclear reprogramming.

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Energy metabolism in human pluripotent stem cells and their differentiated counterparts

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The tumorigenicity of human embryonic and induced pluripotent stem cells

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1 reference

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Mitochondrial rejuvenation after induced pluripotency

1 reference

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Post-transcriptional regulation of the mitochondrial H+-ATP synthase: A key regulator of the metabolic phenotype in cancer

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Human G3BP1 interacts with beta-F1-ATPase mRNA and inhibits its translation.

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A high-throughput respirometric assay for mitochondrial biogenesis and toxicity

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Induced pluripotent stem cells: what lies beyond the paradigm shift

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Down-regulation of mitochondrial ATPase by hypermethylation mechanism in chronic myeloid leukemia is associated with multidrug resistance

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IF1 reprograms energy metabolism and signals the oncogenic phenotype in cancer

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Pharmacological inhibition of fatty acid synthase (FAS): a novel therapeutic approach for breast cancer chemoprevention through its ability to suppress Her-2/neu (erbB-2) oncogene-induced malignant transformation.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3575450

23 June 2018

The soraphens: a family of novel antifungal compounds from Sorangium cellulosum (Myxobacteria). I. Soraphen A1 alpha: fermentation, isolation, biological properties.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3575450

23 June 2018

Metformin lowers the threshold for stress-induced senescence: a role for the microRNA-200 family and miR-205.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3575450

31 August 2018

De novo lipogenesis protects cancer cells from free radicals and chemotherapeutics by promoting membrane lipid saturation

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3575450

31 August 2018

The senescence-related mitochondrial/oxidative stress pathway is repressed in human induced pluripotent stem cells

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3575450

31 August 2018

Selection of cancer cells with repressed mitochondria triggers colon cancer progression

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3575450

31 August 2018

PPARgamma regulates LIF-induced growth and self-renewal of mouse ES cells through Tyk2-Stat3 pathway

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3575450

31 August 2018

Peroxisome proliferator-activated receptor-gamma protects ERBB2-positive breast cancer cells from palmitate toxicity.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3575450

31 August 2018

The effects of BADGE and caffeine on the time-course response of adiponectin and lipid oxidative enzymes in high fat diet-fed C57BL/6J mice: correlation with reduced adiposity and steatosis

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3575450

31 August 2018

Activation of AMP-activated protein kinase reduces hyperglycemia-induced mitochondrial reactive oxygen species production and promotes mitochondrial biogenesis in human umbilical vein endothelial cells

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3575450

31 August 2018

The connections between C75 and obesity drug-target pathways

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3575450

31 August 2018

Why does tumor-associated fatty acid synthase (oncogenic antigen-519) ignore dietary fatty acids?

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3575450

31 August 2018

Fixation Methods for the Study of Lipid Droplets by Immunofluorescence Microscopy

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3575450

31 August 2018

Pluripotency and tumorigenicity.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3575450

31 August 2018

A high glycolytic flux supports the proliferative potential of murine embryonic stem cells

1 reference

https://pubmed.ncbi.nlm.nih.gov/23287468

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Chemical inhibition of acetyl-CoA carboxylase induces growth arrest and cytotoxicity selectively in cancer cells

1 reference

https://pubmed.ncbi.nlm.nih.gov/23287468

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Malonyl-coenzyme-A is a potential mediator of cytotoxicity induced by fatty-acid synthase inhibition in human breast cancer cells and xenografts

1 reference

https://pubmed.ncbi.nlm.nih.gov/23287468

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Identifiers

10.4161/CC.23352

1 reference

Europe PubMed Central

PMC publication ID

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:23287468%20AND%20SRC:MED&resulttype=core&format=json

21 February 2020

PMC publication ID

1 reference

Europe PubMed Central

PMC publication ID

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:23287468%20AND%20SRC:MED&resulttype=core&format=json

21 February 2020

PubMed publication ID

1 reference

Europe PubMed Central

PMC publication ID

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:23287468%20AND%20SRC:MED&resulttype=core&format=json

21 February 2020

ResearchGate publication ID

0 references

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